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And once the virus enters the lungs – hemorrhaging begins immediately in the acinus. A continuous hemorrhage … It takes several hours.
In the blood fibrin is formed, and from it – giolinovaya membrane, resembling a plastic bag. It envelops the acinus, and the person breathes in oxygen, but it is not transferred to the tissues. And people just gasp.
There is a cardio-pulmonary insufficiency and cardiogenic shock. People die of cardiogenic shock.
And there is no pneumonia. Pneumonia – an inflammation, which is treated with antibiotics. Antibiotics cannot help at any stage. There should be absolutely different treatment.
As victims struggled to clear their airways of the bloody froth that poured from their lungs, their bodies started to turn blue from the lack of oxygen, a condition known as violaceous heliotrope cyanosis. “They’re as blue as huckleberries and spitting blood,” one New York City physician told a colleague.
U.S. Army medics noted that this was “not the dusky pallid blueness that one is accustomed to in failing pneumonia, but rather [a] deep blueness…an indigo blue color.” The hue was so dark that one physician confessed that “it is hard to distinguish the colored men from the white.”
“It is only a matter of a few hours then until death comes,” recalled another physician, “and it is simply a struggle for air until they suffocate.” They drowned in their own bloody secretions.
“It wasn’t always that quick, either,” one historian adds. “And along the way, you had symptoms like fingers and genitals turning black, and people reporting being able to literally smell the body decaying before the patient died.” “When you’re ill like that you don’t care,” recalls one flu survivor, now 100 years old. “You don’t care if you live or die.”
Signs and symptoms of bubonic plague include:
■Buboes — swollen, painful, warm lymph nodes
■Sudden onset of fever and chills
■Fatigue or malaise
Septicemic plague occurs when plague bacteria multiply in your bloodstream. If septicemic plague occurs as a complication of bubonic plague, buboes may be present.
Signs and symptoms include:
■Fever and chills
■Abdominal pain, diarrhea and vomiting
■Bleeding from your mouth, nose or rectum, or under your skin
■Blackening and death of tissue (gangrene) in your extremities, most commonly your fingers, toes and nose
Pneumonic plague — which can occur as a complication of another type of plague or by inhaling infectious droplets coughed into the air by a person or animal — is the least common form of plague. But it's also the most rapidly fatal. Early signs and symptoms, which generally occur within a few hours to a few days after inhaling contaminated droplets, include:
■Signs of pneumonia, including chest pain, difficulty breathing and a cough with bloody sputum
■Nausea and vomiting
Pneumonic plague progresses rapidly and may cause respiratory failure and shock within two days of infection. If antibiotic treatment isn't initiated within a day after signs and symptoms first appear, the infection is likely to be fatal.
Ukrainian News Agency "Fraza" reported that, according to informed sources, "it has been confirmed 100 % Pneumonic Plague in Ukraine".
The Agency asserts that "the head physician of the medical institutions has sent out an informal disposal - not to sow panic, to refute the information about the plague, and to speak only of swine influenza".
CDC Estimates of 2009 H1N1 Influenza Cases, Hospitalizations and Deaths in the United States, April – October 17, 2009•CDC estimates that between 14 million and 34 million cases of 2009 H1N1 occurred between April and October 17, 2009. The mid-level in this range is about 22 million people infected with 2009 H1N1.
•CDC estimates that between about 63,000 and 153,000 2009 H1N1-related hospitalizations occurred between April and October 17, 2009. The mid-level in this range is about 98,000 H1N1-related hospitalizations.
•CDC estimates that between about 2,500 and 6,000 2009 H1N1-related deaths occurred between April and October 17, 2009. The mid-level in this range is about 3,900 2009 H1N1-related deaths.
By the early 1990s, 75 years of research had failed to answer a most basic question about the 1918 pandemic: why was it so fatal? No virus from 1918 had been isolated, but all of its apparent descendants caused substantially milder human disease. Moreover, examination of mortality data from the 1920s suggests that within a few years after 1918, influenza epidemics had settled into a pattern of annual epidemicity associated with strain drifting and substantially lowered death rates. Did some critical viral genetic event produce a 1918 virus of remarkable pathogenicity and then other critical genetic event occur soon after the 1918 pandemic to produce an attenuated H1N1 virus?
In 1995, a scientific team identified archival influenza autopsy materials collected in the autumn of 1918 and began the slow process of sequencing small viral RNA fragments to determine the genomic structure of the causative influenza virus (10). These efforts have now determined the complete genomic sequence of 1 virus and partial sequences from 4 others. The primary data from the above studies (11–17) and a number of reviews covering different aspects of the 1918 pandemic have recently been published (18–20) and confirm that the 1918 virus is the likely ancestor of all 4 of the human and swine H1N1 and H3N2 lineages, as well as the "extinct" H2N2 lineage. No known mutations correlated with high pathogenicity in other human or animal influenza viruses have been found in the 1918 genome, but ongoing studies to map virulence factors are yielding interesting results. The 1918 sequence data, however, leave unanswered questions about the origin of the virus (19) and about the epidemiology of the pandemic.
At the beginning of other "off season" influenza pandemics, successive distinct waves within a year have not been reported. The 1889 pandemic, for example, began in the late spring of 1889 and took several months to spread throughout the world, peaking in northern Europe and the United States late in 1889 or early in 1890. The second recurrence peaked in late spring 1891 (more than a year after the first pandemic appearance) and the third in early 1892 (21). As was true for the 1918 pandemic, the second 1891 recurrence produced of the most deaths. The 3 recurrences in 1889–1892, however, were spread over >3 years, in contrast to 1918–1919, when the sequential waves seen in individual countries were typically compressed into ≈8–9 months.
One theory that may partially explain these findings is that the 1918 virus had an intrinsically high virulence, tempered only in those patients who had been born before 1889, e.g., because of exposure to a then-circulating virus capable of providing partial immunoprotection against the 1918 virus strain only in persons old enough (>35 years) to have been infected during that prior era (35). But this theory would present an additional paradox: an obscure precursor virus that left no detectable trace today would have had to have appeared and disappeared before 1889 and then reappeared more than 3 decades later.
Could a 1918-like Pandemic Appear Again? If So, What Could We Do About It?
In its disease course and pathologic features, the 1918 pandemic was different in degree, but not in kind, from previous and subsequent pandemics. Despite the extraordinary number of global deaths, most influenza cases in 1918 (>95% in most locales in industrialized nations) were mild and essentially indistinguishable from influenza cases today. Furthermore, laboratory experiments with recombinant influenza viruses containing genes from the 1918 virus suggest that the 1918 and 1918-like viruses would be as sensitive as other typical virus strains to the Food and Drug Administration–approved antiinfluenza drugs rimantadine and oseltamivir.
Originally posted by JJay55
Originally posted by not_just_paranoid
What facts do you have to base this assumption? I have the UN Who's reports of 1972 telling how to commit world genocide, and they have followed this plan to the letter T.
Think about it now, if we have too many people and not enough food, how are 550,000 soliders, national guard, reserves, police, sheriffs, going to stop 300,000 million people at 5,450 to 1 odds? Bio-weapons is the answer for the rich power elite. They have been planning this from day one of the League of Nations to the United Nations of today. Of the 194 countries that signed onto the WHO, if a stage six pandemic is announced, those national sovereign countries give up their sovereignty to the WHO. We are talking about the One World Government or New World Order. You my dear have not done your homework.
Originally posted by Kailassa
Originally posted by Asherah
Originally posted by Kailassa
Ginger is a great anti-inflammatory. Besides, they go beautifully with brandy. Prednisolone is another way to treat it, under medical supervision.
Alcohol, once one is infected with swine flu, avian flu or sars, is an absolute no-no. It worsens inflammation.
Wouldn't taking ibuprofen generate the same anti inflammatory affect?
I don't see the point in taking a drug when a common food serves the same purpose.
Ginger has no bad side effects. As well as being an anti-inflammatory it is safe to use long term, it is better than any anti-nausea drug, it gives one an appetite, it's readily available, cheap and it tastes good.
Originally posted by really
... I do have a friend who caught the flu. She dowsed herself in different homeopathic remedies and started feeling better. ... She tested positive for swine flu. So, she took herself to her own doctor who confirmed this. He also found that she had pneumonia.