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Originally posted by invernal
How did they think it wouldn't be transparent, or at least odd to us that they predicted this so accurately?
Originally posted by JJay55
Originally posted by apacheman
Just got a report a few minutes ago from an ER nurse friend in Phoenix.
She reports no cases of hemorrhagic flu there yet, BUT....
She also reported that two pregnant women were recently brought in with flu symptoms and both died within hours of arrival, losing both mother and child. No autopsies yet, and no one knows what exactly killed them.
She'll be giving me updates every day or so, so I'll let you guys know the moment I hear anything worth reporting. I'm trying to establish a network of ER personnel to report to me when they can. I strongly suggest that if you know any ER people you do the same and we collate all reports here.
Swine flu causes spontaneous abortion.
The receptor binding domain (RBD) for the Ukraine H1N1 mutation, D225G, has been linked to fatal cases of swine flu infection. Demographic and genetic analysis for the H1N1 mutation taking place in the Ukraine virus has been published by Mill Hill, the London laboratory that performed testing on samples from the Ukraine. D225G is the same RBD that was found in the virus that caused the 1918 Spanish flu outbreak.
H1N1 mutation fatalities
According to this analysis of the Mill Hill lab information, each of the fatal cases of flu from the Ukraine virus contained this marker. D225G is the name for the receptor binding domain of this particular virus. A virus attaches to charged molecules on host cells, but each type of virus attaches to a different type of molecule, in a different way. The H1N1 mutation found in the Ukraine attaches in the same place, in the same way, as the Spanish flu virus from 1918.
Detection of the H1N1 mutation
While positive results for the H1N1 virus with D225G are not widespread, this may be the result of the methods of testing. Tests for swine flu typically consist of a nasal swab, which is unlikely to pick up this mutated virus, due to its location. The H1N1 mutation that is causing fatalities in the Ukraine and Norway affects the respiratory system, and is found deep in the lungs.
Originally posted by marg6043
reply to post by Kailassa
The scaremongering about the swine flu is getting people all looking for anything that happens around and weird by hyper up news of deaths to think that is some mutation into a monster that is going to kill us all.
First of all swine flu is not the 1918 flu that cause so many death due to many environmental and human conditions at the time, 1918 was more linked to symptoms of Ebola, if any case of Ebola symptoms appears in the US then the government would not be so lie back as its right now, Ebola is fatal.
Swine flu has been around for decades infecting people without even been acknowledge until 1976, when it was time for money making by the big pharma and cash on it.
By CDC statistics swine flu has infected over 22 million of people in the US, and we don't have mass graves to call it a pandemic, but the CDC had to go back to April to scrape for pneumonia death cases to account for the almost 5 thousand deaths so far.
That is why I keep telling that what is killing is the pneumonia that in most cases is going untreated because you can get pneumonia with or without swine flu and is growing problem in the US.
Statistics show that 5 to 10 million people contract pneumonia in the nation every year without swine flu to help and seasonal flu side effects also affect pneumonia.
With 50 to 70 thousand deaths a year alone.
Is very clear that Pneumonia side effects if untreated causes swelling of the brain and the hart, causes edema and lungs to fill with phlegm in chronic untreated cases the lungs will fill with pus and abscess that will bleed into the lungs.
And that is what I have researched so far.
Originally posted by downtown436
-Based on what has happened to Moshe, his info was correct. A mutated swine flu, that is the 1918 strain has been released in the Ukraine, and now has spread across the globe. It was probably in vaccines, and sprayed by aircraft. (yes it is a bio attack).
Originally posted by invernal
Originally posted by Kailassa
I don't see the point in taking a drug when a common food serves the same purpose.
Ginger has no bad side effects. As well as being an anti-inflammatory it is safe to use long term, it is better than any anti-nausea drug, it gives one an appetite, it's readily available, cheap and it tastes good.
Some people are allergic to the food "medicine", including even ginger (or the whole ginger family which includes a bunch of spices like turmeric and cardamom, too).
Bromelain (pineapple enzyme) is an anti-inflammatory, which I've read can even work better than ibuprofen for some issues (*don't* know about with H1N1). However, people with latex allergies may be allergic to that, too (as well as kiwi, avocados, bananas, and who knows what else in that allergy cross-reaction group).
There is also a caution with bromelain, as it can increase the heartrate. It can also make anything you take with it more bio-available or potent, meaning you could also increase potentially dangerous side effects.
So, just because it's natural doesn't mean it's ideal for a given person.
I still prefer natural, but I myself took tylenol when I got my H1N1 fever. It was the only drug (granted, OTC) that I took, because I didn't know what else to take and the fever isn't something I play around with.
Originally posted by marg6043
I am very confidence with all the information I have gathered on the influenza to say that things will calm down and die eventually.
Evidence on H1N1 D225G in Lung Cases in Norway and Ukraine Recombinomics Commentary 11:29 November 21, 2009 The WHO said the mutation does not appear to spread and the public health significance of the finding is unclear.
"Although further investigation is under way, no evidence currently suggests that these mutations are leading to an unusual increase in the number of H1N1 infections or a greater number of severe or fatal cases," the agency said.
The above WHO comments on the receptor binding domain change D225G (cited as position 222 in some reports using H1 numbering) is curious. Although WHO claimed that there were no significant changes in the sequences from Ukraine, none of the Ukraine situation updates excluded a receptor binding domain change, and when 10 HA sequences were published at GISAID by WHO regional lab Mill Hill, there was clear evidence of an association of D225G with fatal cases. Of the 10 sequences released, four were listed as deceased, and all four had D225G. None of the six samples which came from patients that were labeled with age and gender but had no deceased designation had D225G. Moreover, three of the four samples from deceased patients were lung samples and all three lung samples had D225G. The samples which had closely related sequences but lacking D225G were predominantly nasal washes, which also presented concerns that the swine H1N1 was differentially detected, due in part to changes in receptor binding specificity.
This concern was based on published data on sequences from samples from 1918 patients. In 1918 the same change occurred. Most samples had a D at position 225, while two samples had D225G (from 1918 and 1919). The tested sample with D225G had a different binding specificity, which would be expected to show differential binding to various tissue types.
Moreover, the D225G identified from Ukraine was in both of the hard hit areas, Ternopil and Lviv. Two samples from each area were positive, indicating the change had spread. Ukraine has already acknowledged over 350 dead patients and initial reports described hemorrhagic lungs that were totally destroyed. This destruction was in multiple media reports as well as an agency report describing 90 fatal cases.
Moreover, the evidence was not limited to Ukraine. Earlier samples from lung tissues from deceased patients in Sao Paulo also had D225G, as did patients in multiple countries. Most cases were not described in detail, but the case from China was from the first severe case in Zheijiang Province and although the patient survived, she was hospitalized for several weeks.
In addition, the presence of the same change on multiple backgrounds indicated the polymorphism was being acquired via recombination. The sudden appearance of the same polymorphisms on multiple backgrounds has been described previously, including a silent change in H5N1 and H274Y in seasonal flu. These changes allowed for the prediction of D225G in the Ukraine patients prior to release of the sequences.
The above comments were made after D225G was found in samples from deceased or severe cases in Norway, which provided further evidence of spread and association with lung samples.
Thus, there was and is ample evidence of D225G in severe and fatal cases. Like 1918, it is not in all samples from fatal cases, and as with all infectious disease, not all infections are fatal. If the initial dose is low, or the hosts mounts and effective early defense, the clinical course may be mild, as has been seen with virtually all influenza infections, including H5N1 infections such as those in Egypt.
Thus, the presence of the 1918 receptor binding domain change in an H1N1 swine virus that has jumped to humans is cause for concern, and a more comprehensive survey of lung samples is useful, as well as release of new receptor binding data.
The WHO added, "The outbreak in Ukraine may be indicative of how the virus can behave in the northern hemisphere during the winter season, particularly in health care settings typically found in Eastern Europe. Given the potential significance of this outbreak as an early warning signal, WHO commends the government of Ukraine for its transparent reporting and open sharing of samples." The samples have been sent to the WHO Mill Hill Influenza Reference Lab in London, not exactly inspiring confidence in a scientifically honest report given the record of UK health authorities in manipulating data to please the vaccine giants like GlaxoSmithKline. As of this writing, bizarre enough the WHO has yet to utter a single word of the test results at Mill Hill.