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originally posted by: makemap
Don't listen that you need more doctors in one area, its a freaking trap. All the Doctors are just going to be murdered by the one mad person.
#1 Here is how to get rid of Ebola, shut border.
#2 Find the Source (which no one does anymore)
#3 Cremate/Disinfect starting from the Source.
#4 Recycle Water to be clean again.
originally posted by: crazyewok
originally posted by: ATF1886
S&F as always yea im stocking up on nano silver vitamin c oregeno oil
Dont buy the snake oil BS
That wont but a dent in a Ebola infection.
originally posted by: vaibow123
I think what frightens me, is the quiet time/incubation period.
If it can be contracted and carried for a few weeks before signs even show, then this is where the repercussions are serious - as everyone is saying.
If a terrorist group, whether it be home grown or over seas really wanted to do some damage - all they need is one person to come in contact with an infected individual then spend the next fortnight on as much public transport/places as they can - that would do more damage than any suicide bombing. Frightening.
Boneheaded ISIS Threat: We Will Infect U.S. With Ebola
Here's one terror threat you can ignore
….Unless ISIS recruits an expert from the Centers for Disease Control, we’re fine.
originally posted by: Wirral Bagpuss
Now might be a good time to watch BBC Survivors and Twelve Monkeys. They both dealt with the threat of a plague/disease that would wipe out mankind. Survivors is excellent. It shows what happens when most of the world's population is dead and global population is only a few million or so. No government, nothing.
I always thought it would be a reoccurrence of the 1918 flu that would be the end of us. Now it seems Ebola could well become the most serious threat to date. Heck if it came to it I would stock up on food and water etc and isolate myself if it became an epidemic here. I would either go and hide in the Scottish Highlands and islands or the most rural part of Wales. I do have a bolt away in a very remote place that I can get to for when things get out of control.
originally posted by: soficrow
a reply to: Char-Lee
We should be afraid. We are contaminating our planet with nearly 100,000 synthetic chemicals that can combine into an infinite number of new never-seen-before chemical compounds. We're messing with the nano-structures of our world along with molecules. Of course microbes, viruses and fungi are mutating - they need to adapt and evolve to survive in the new environment we have created. and fyi - so do we - this is NOT the Earth we evolved to live and thrive on. ...A big part of the evolutionary process is disease. Sucks, but true.
I agree on the big picture..
No. That's an urban myth. Ebola is not infectious until symptoms appear -
Viral shedding refers to the expulsion and release of virus progeny following successful reproduction during a host-cell infection. Once replication has been completed and the host cell is exhausted of all resources in making viral progeny, the viruses may begin to leave the cell by several methods.
The term is used to refer to shedding from a single cell, shedding from one part of the body into another part of the body, and shedding from bodies into the environment where the viruses may infect other bodies.
Assessment of the Risk of Ebola Virus Transmission from Bodily Fluids and Fomites
....the shedding of EBOV in saliva corresponded almost exactly to the period of viremia, with the last positive saliva specimen noted at day 8 after disease onset.
....The isolation of virus from breast milk in one case even after clearance from the blood suggests that transmission may occur even during convalescence. It is possible that the mammary gland, like the gonads  and chambers of the eye [13, 14], is an immunologically protected site in which clearance of virus is delayed. However, we cannot rule out that the finding simply represents residual EBOV secreted into the milk during the period of viremia but not expressed until some days later...
....The isolation of EBOV from semen 40 days after the onset of illness underscores the risk of sexual transmission of the filoviruses during convalescence. Zaire EBOV has been detected in the semen of convalescent patients by virus isolation (82 days) and RT-PCR (91 days) after disease onset [5, 14].
....The absence of EBOV infection in multiple tested urine specimens suggests that the virus may not be efficiently filtered in the kidney. Consequently, exposure to urine appears to be of low risk during both acute illness and convalescence. The absence of EBOV in the urine, low prevalence on the skin, and rapid clearance from the saliva in surviving patients provides some reassurance that the risk of secondary transmission from casual contacts, fomites, or the sharing of toilet facilities in the home after discharge from the hospital is minimal. This conclusion is supported by previous empirical observations [5, 6].
....Other than in samples grossly contaminated with blood, EBOV was not found by any method on environmental surfaces and by RT-PCR on the skin of only 1 patient. These results suggest that environmental contamination and fomites are not frequent modes of transmission, at least in an isolation ward.
....Taken together, our results support the conventional assumptions and field observations that most EBOV transmission comes from direct contact with blood or bodily fluids of an infected patient during the acute phase of illness. The risk of casual contacts with the skin, such as shaking hands, is likely to be low. Environmental contamination and fomites do not appear to pose a significant risk when currently recommended infection control guidelines for the viral hemorrhagic fevers are followed.
Replication, Pathogenicity, Shedding, and Transmission of Zaire ebolavirus in Pigs
....Results. Following mucosal exposure, pigs replicated ZEBOV to high titers (reaching 107 median tissue culture infective doses/mL), mainly in the respiratory tract, and developed severe lung pathology. Shedding from the oronasal mucosa was detected for up to 14 days after infection, and transmission was confirmed in all naive pigs cohabiting with inoculated animals.
Conclusions. These results shed light on the susceptibility of pigs to ZEBOV infection and identify an unexpected site of virus amplification and shedding linked to transmission of infectious virus.
.........There is increasing experimental evidence indicating that the Ebola virus glycoprotein can mediate entry from the apical side into intact airway epithelia of mouse, nonhuman primate, or human origin [28, 30–32]. The presence of Ebola antigens was also detected in the respiratory mucosa, alveoli, and pulmonary lymphatic tissue of nonhuman primates following aerosolized Ebola challenge, demonstrating that the virus can infect nonhuman primates through mucosal exposure with ebolavirus .
.....These studies underline some differences in the pathology induced by ZEBOV in pigs, compared with nonhuman primates and humans. In contrast to the severe systemic syndrome often leading to shock and death in primates, pigs developed a respiratory syndrome that could be mistaken for other porcine respiratory diseases.
....These data also have implications for the management of human outbreaks following accidental or hypothetically intentional exposure of pigs to Ebola virus.(!)