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originally posted by: snowspirit
www.heretohelp.bc.ca...
A Canadian study.
What researchers have learned from these studies is that cannabis can potentially cause psychosis, but only in a select group of people who are naturally vulnerable.
originally posted by: snowspirit
Most people's paranoia comes from worrying if other people know that you're high. Once that stigma of "doing something illegal" is gone, so is the paranoia.
originally posted by: theabsolutetruth
a reply to: OneManArmy
Verbal attacks, presumptions and accusations are not rational questions.
originally posted by: theabsolutetruth
a reply to: OneManArmy
Verbal attacks, presumptions and accusations are not rational questions.
originally posted by: theabsolutetruth
a reply to: OneManArmy
Verbal attacks, presumptions and accusations are not rational questions.
so all the idiots and their irrational vitriolic attacks are just proving the research correct
I put it down to the enormous popularity of coc aine as a recreational drug.
This is another study that is trying to correlate cannabis use to psychosis without taking into consideration that MAYBE people who are prone to psychosis seek out cannabis (and other drugs) as a form of self-medication.
Bullcrap. Here is a great refutation of your study...
"The observed tendency for cannabis use to precede or coincide with rather than follow mania symptoms, and the more specific association between cannabis use and new onset manic symptoms, suggests potential causal influences from cannabis use to the development of mania. It is a significant link."
Dr Marwaha also said the review suggested that cannabis use significantly worsened mania symptoms in people who had previously been diagnosed with bipolar disorder.
He added: "There are limited studies addressing the association of cannabis use and manic symptoms, which suggests this is a relatively neglected clinical issue. However our review suggests that cannabis use is a major clinical problem occurring early in the evolving course of bipolar disorder..." Source
How The Lancet handles your paper
Peer review
Every Article, Case Report, Hypothesis, Seminar, and Review published in The Lancet has been peer reviewed. Occasional contributions (eg, Essays) are accepted without peer review.
On submission to The Lancet, your report will first be read by one or more of the journal's staff of physicians and scientists. Our acceptance rate overall is about 5% and it is an important feature of our selection process that many papers are turned away on the basis of in-house assessment alone. That decision will be communicated quickly.
Research papers and most other types of paper that receive positive in-house reviews are followed by peer review by at least three reviewers. You will receive notification of which editor is handling the peer review of your paper. If reviews are encouraging, and the editorial consensus is also favourable, then we seek statistical advice where appropriate. Source
originally posted by: Astyanax
a reply to: tothetenthpower
Since there appears to be some question about whether the paper posted by the OP was peer-reviewed or not,
Tuesday, the British Medical Journal published the second of its three-part series claiming that the 1998 study in The Lancet that sparked fears that the measles-mumps-rubella vaccine could cause autism was the result of deliberate fraud and profiteering.
A new investigation into Dr. Andrew Wakefield’s hotly contested MMR-autism study concluded there was no fraud committed by Dr. Wakefield
At least 28 studies from around the world support Dr. Wakefield’s controversial findings; Dr. Wakefield has also published dozens of peer-reviewed papers looking at the mechanism and cause of inflammatory bowel disease, and has extensively investigated the brain-bowel connection in the context of children with developmental disorders such as autism.
Growing research is now supporting the link between gastrointestinal disturbances and autism; it could be that if your child has suboptimal gut flora, vaccines can become the proverbial "last straw"—the trigger that sends his/her immune system over the edge into the development of chronic heath problems
We investigated the association between cannabis use and risk of psychosis by analysing the expression of psychotic experiences. Psychotic experiences share many characteristics with clinically relevant psychosis, such as demographic, environmental, and genetic risks, and are thought to represent a behavioural marker for psychosis liability.18 31 32 Psychotic experiences are a common and generally transitory phenomenon in the general population, that, nevertheless, might become abnormally persistent and progress to clinical psychotic disorder if combined with exposure to environmental risks.19 33 34 Our study confirmed cannabis as an environmental risk factor, impacting on risk of psychosis by increasing the risk of incident psychotic experiences, and, if use continues over time, increasing the risk of persistent psychotic experiences.
The finding that longer exposure to cannabis was associated with greater risk for persistence of psychotic experiences is in line with an earlier study showing that continued cannabis use over time increases the risk for psychosis in a dose-response fashion.5 This is also in agreement with the hypothesis that a process of sensitisation might underlie emergence and persistence of psychotic experiences35 as an indicator of liability to psychotic disorder.18 31 Sensitisation refers to the phenomenon that repeated exposure to an (environmental) stressor leads to progressively greater responses over time.35 36 37 38 In rats, repeated exposure to THC (delta-9-tetrahydrocannabinol, the main psychoactive component of cannabis) induces behavioural sensitisation: rats pre-treated with increasing doses of THC show greater behavioural (locomotor) responses to a THC challenge after a 14 day washout period than THC naive rats.39 40 In humans, however, direct evidence for cannabis sensitisation is lacking.
As our study showed that the risk of persistent psychotic experiences increases with longer periods of cannabis exposure, we suggest that a process of sensitisation underlies the association between cannabis and psychosis.32