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The FDA has just notified one pharmacy that it will no longer be allowed to manufacture or distribute injectable vitamin C—despite its remarkable power to heal conditions that conventional medicine can’t touch...
...it now insists on wiping out one of the best potential treatments for these conditions and for certain cancers as well. And why is this being done? What possible rationale is offered? Because it’s dangerous? No. Because it can’t be patented...
Each of us reading this should think, “Intravenous C could someday save my life.” Dr. Jonathan Collin, editor of the Townsend Letter, discusses the case of a man in New Zealand who nearly died from swine flu. After developing a severe fever and upper respiratory infection, his condition deteriorated and he became comatose. Eventually even a ventilator was insufficient to keep him breathing because his lungs were so compromised by pulmonary edema. After weeks of heroic intervention, doctors decided there was no chance of survival and nothing further should be done for him.
The family asked the hospital to administer intravenous vitamin C. After much disagreement, the hospital gave him 25 grams of vitamin C every 6 hours. There was so much improvement over the next two days that the hospital decided to reinstate his intensive care—but they discontinued the vitamin C, saying that he had improved only because they had rolled him onto his side or his stomach instead of keeping him on his back! Not surprisingly, his condition once again deteriorated.
The family moved him to another facility that reluctantly allowed the IV vitamin C (albeit at a lower dose), and his lung function gradually improved. He came out of coma after four weeks, and after taking vitamin C orally, he gradually improved enough to be discharged. One year later, he was back to flying his plane and surveying his farm in New Zealand.
Originally posted by burntheships
We should have access to pharmacuetical grade Vitamin C as a healing agent without FDA control,
Vitamin C is not a drug, see then why is the FDA pawing at it?
Vitamins are not individual molecular compounds. Vitamins are biological complexes. They are multi-step biochemical interactions whose action is dependent upon a number of variables within the biological terrain. Vitamin activity only takes place when all conditions are met within that environ-ment, and when all co-factors and components of the entire vitamin complex are present and working together. Vitamin activity is even more than the sum of all those parts; it also involves timing.
Around 1912, Kazimierz Funk (1884-1967) introduced the term “Vitamines” to define compounds whose dietary deficiency caused severe diseases in humans. In Funk’s list of such nutritional factors, the name “Vitamin C” was given to the factor counteracting the syndrome known as scurvy. At the time, Vitamin C was known to occur in fresh fruits and vegetables, but its molecular identity was still a mystery.
Only a few years later, Albert von Szent Gyorgyi (1893-1985) was working at the characterization of a six-carbon sugar he had obtained in crystalline form from both animal and plant tissues. Initially, he used the funny names “ignose” and “godnose” (from “God knows”) to indicate this molecule of unknown biological function, then he switched to a more neutral “hexuronic acid”.
In 1929, Szent Gyorgyi and Walter Haworth (1883-1950) could eventually identify the unknown sugar as the anti-scurvy factor Vitamin C and consequently named it “ascorbic acid”, a discovery that earned them the 1937 Nobel Prize for Medicine and Chemistry, respectively. Later on, ascorbic acid was identified as an essential factor in collagen synthesis, which accounts for its effect in scurvy prevention and cure.
Different collagen types are the most abundant proteins of the extracellular matrix, and have a basic role in strengthening mechanical tissues. In the absence of ascorbic acid, collagen fibrils are not properly assembled. Between 1950 and 1960, the molecular mechanism underlying Vitamin C protective action was discovered. Proper collagen assembly only occurs when some proline and lysine residues in the protein are selectively hydroxylated to hydroxyproline and hydroxylysine, two reactions catalyzed by the ascorbic acid-dependent enzymes prolyl- and lysyl hydroxylase (De Tullio 2004)