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Ketone bodies are three different water-soluble, biochemicals that are produced by the liver from fatty acids during periods of low food intake (fasting) and starvation for cells of the body to use as energy instead of glucose. Two of the three are used as a source of energy in the heart and brain while the third (acetone) is a degradation breakdown product of acetoacetic acid. Radioactive tracing of acetone determines that between 2% and 30% is excreted from the body. Ketone bodies are picked up by cells and converted back into acetyl-CoA which then enters the citric acid cycle and electron transport chain for energy. In the brain ketone bodies are also used to make Acetyl CoA into long chain fatty acids because long chain fatty acids cannot pass through the blood brain barrier. The liver breaks down protein to produce glucose during starvation for the very few glucose obligate cells that cannot use ketone bodies. In the brain, ketone bodies are a vital source of energy during fasting or strenuous exercise. Although termed "bodies", they are molecules, not particles.
The three endogenous ketone bodies are acetone, acetoacetic acid, and beta-hydroxybutyric acid. Other ketone bodies such as beta-ketopentanoate and beta-hydroxypentanoate may be created as a result of the metabolism of synthetic triglycerides such as triheptanoin.
reply to post by Ektar
Do what you feel you will be able to follow through with. If low carb/paleo eating isn't for you, then you won't stick with it. Just do what feels right (even if that means carrying a few extra pounds).
But it would be interesting to see a new Reality TV show challenge how the old way
of eating verses how people eat today with all the processed food!
I am afraid to do the fats because I think I'm fat
Specifically, we have examined the interaction between the APOA5-1131T>C and 56C>G (S19W) polymorphisms and the macronutrient intake (total fat, carbohydrate, and protein) in their relation to the body mass index (BMI) and obesity risk in 1,073 men and 1,207 women participating in the Framingham Offspring Study. We found a consistent and statistically significant interaction between the -1131T>C single-nucleotide polymorphism (SNP; but not the 56C>G) and total fat intake for BMI.
In conclusion, the APOA5-1131T>C SNP, which is present in approximately 13% of this population, modulates the effect of fat intake on BMI and obesity risk in both men and women.