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Posted by Katelyn Sabochik on July 14, 2010 at 02:26 PM EDT
The National HIV/AIDS strategy focuses on three major goals: reducing the number of new infections, increasing access to care and optimizing health outcomes for people living with HIV and AIDS, and reducing health-related disparities.
President Obama committed to developing a National HIV/AIDS Strategy with three primary goals:
1. reducing the number of people who become infected with HIV,
2. increasing access to care and optimizing health outcomes for people living with HIV, and
3. reducing HIV-related health disparities.
We must also move away from thinking that one approach to HIV prevention will work, whether it is condoms, pills, or information. Instead, we need to develop, evaluate, and implement effective prevention strategies and combinations of approaches including efforts such as expanded HIV testing (since people who know their status are less likely to transmit HIV), education and support to encourage people to reduce risky behaviors, the strategic use of medications and biomedical interventions (which have allowed us, for example, to nearly eliminate HIV transmission to newborns), the development of vaccines and microbicides, and the expansion of evidence-based mental health and substance abuse prevention and treatment programs.
Reducing HIV–Related Health Disparities
The stigma associated with HIV remains extremely high and fear of discrimination causes some Americans to avoid learning their HIV status, disclosing their status, or accessing medical care. Data indicate that HIV disproportionately affects the most vulnerable in our society—those Americans who have less access to prevention and treatment services and, as a result, often have poorer health outcomes. Further, in some heavily affected communities, HIV may not be viewed as a primary concern, such as in communities experiencing problems with crime, unemployment, lack of housing, and other pressing issues. Therefore, to successfully address HIV, we need more and better community–level approaches that integrate HIV prevention and care with more comprehensive responses to social service needs. Key steps for the public and private sector to take to reduce HIV-related health disparities are:
* Reduce HIV-related mortality in communities at high risk for HIV infection.
* Adopt community-level approaches to reduce HIV infection in high-risk communities.
* Reduce stigma and discrimination against people living with HIV.
Originally posted by pikypiky
See? First we get the health care insurance reform and now infectious diseases through the IV. This will teach people to take better care of the themselves and watch out not to hurt others so they too could avoid hospitalization and blood transfusion. I wonder how the vampyres would respond to the contamination of their `blood supply`?
Originally posted by halfoldman
reply to post by halfoldman
Looking again at the law, it's actually very dangerous.
It's telling the careless idiot, the fatalist or the sociopath:
"Don't test - what you don't know can't legally hurt you!"
Just assume you're HIV-negative and tell everyone that you are!
What kind of message is that?
HIV does not cause AIDS, it is just a harmless passenger virus, that's the claim of Duesberg and colleagues. The WHO (World Health Organization) estimates that 34.3 million are HIV-positive worldwide in 2000, yet only 1.4% developed AIDS. Similarly, in 1985, only 1.2% of the 1 million US citizens with HIV developed AIDS.
Peter Duesberg was, and still is, professor of molecular biology at the University of California at Berkeley, member of the National Academy of Sciences and recipient of a 1985 Outstanding Investigative Grant from the National Institutes of Health. He was tipped as a Nobel candidate for his work on viral oncogenes (genes causing cancer).
But all that came to a crashing end in 1987, when he published a paper  claiming that HIV did not cause AIDS, contrary to what the scientific community had come to believe to this day (Box 1), but was instead the result of drug use. He soon lost all his research grants, but that has not silenced him.
Ironically, Duesberg's hypothesis was generally held before the idea that HIV caused AIDS became accepted (see Box 2).
A brief history of HIV-AIDS hypothesis In 1981, a new epidemic began to strike male homosexuals and intravenous drug users in the United States and Europe. The US Centers for Disease Control (CDC) termed the epidemic, AIDS, for acquired immunodeficiency syndrome.
Between 1981 and 1984, leading researchers, including those from CDC proposed that recreational drug use was the cause of AIDS.
But in 1984, the US government researchers proposed that a virus, now termed human immunodeficiency virus (HIV), is the cause of the epidemic in US and Europe, and also in Africa. This hypothesis - HIV causes AIDS gained instant acceptance within the scientific community.
The report which received most attention at the VIII International Conference on AIDS was a presentation describing several cases of severe immuno-deficiency in persons without detectable HIV at a "Recent Reports" session at the conference, Dr. Jeffrey Laurence of Cornell Medical Center reported five cases of immune suppression characterized by low T4 cell counts, opportunistic infections, like CMV colitis, PCP and KS. Some patients had risk factors for HIV-1 infection yet none had any evidence of HIV-1 or HIV-2. Dr. James Curran, Director of AIDS at the Centers for Disease Control (CDC), reported six additional cases which had been reported to the CDC in the past years. The agency chose not to report these cases and received severe criticism by many researchers for that decision. Other researchers reported similar HlV-negative cases of immune-suppression, including Dr. David Ho with 11 cases of patients. These were mostly gay men with low T4 cell counts, three of whom had OIs. In addition, Luc Montagnier, the co-discover of HIV-1, reported experience with a similar case. He claimed to have found HIV in the urine of a patient whose blood had no traces of the virus after PCR analysis.
Idiopathic CD4+ T-lymphocytopenia, or ICL, is an immunodeficiency syndrome in which human immunodeficiency virus, or HIV, cannot be detected. Because HIV is the causative agent of acquired immune deficiency syndrome (AIDS), ICL can be referred to as Non-HIV AIDS. As in AIDS patients, Non-HIV AIDS patients exhibit reduced numbers of CD4+ T-lymphocytes, and many Non-HIV AIDS patients have developed the opportunistic infections or otherwise rare cancers associated with AIDS.
Corticosteroids, Illicit Drugs, and Malnutrition to the Pathogenesis of AIDS
Review of the medical literature concerning the causes and the pathogenesis of AIDS worldwide, revealed the following facts:
1. AIDS in drug users and homosexuals in the USA and Europe is probably caused by the heavy ancillary use of glucocorticoids and other immunosuppressive agents to medically treat the wide range of the chronic serious illnesses.
2. AIDS in hemophiliacs is clearly related to the use of corticosteroids and other immunosuppressive agents to prevent the development of antibodies for factors VIII and IX and to treat chronic illnesses.
3. AIDS in people receiving blood and/or tissue is related to the use of glucocorticoids to prevent reactions of transfusion and tissue rejection.
4. AIDS in infants and children is probably caused by their exposure to drugs and corticosteroids in utero and their exposure to corticosteroids after birth used to treat their chronic illnesses.
5. AIDS in Africa is caused by malnutrition, release of endogenous cortisol, and by opportunistic diseases. Atrophy in the lymphoid tissue has been observed in HIV-negative people suffering from malnutrition.
6. Damage to the immune system is rapidly reversible after removal of the true insulting agent or treatment of the true causes in both HIV-positive and HIV-negative AIDS patients.
7. Kaposi’s sarcoma (KS) and lymphoma are probably induced by the use of steroids and drugs, and the release of endogenous cortisol. 8) HIV appears to be a harmless virus both in the in vivo and the in vitro settings.
8. The uses of glucocorticoids, AZT, and protease inhibitors to treat AIDS are contraindicated.
The following case history was the spark that ignited this in-depth investigation of the causes and pathogenesis of acquired immune deficiency syndrome (AIDS). A 60 year-old-white male, HIV-negative, developed Acquired Immune Deficiency Syndrome (AIDS) following treatment with a two month course of prednisone (60 mg per day) and a two week course of azathioprine (50-100 mg per day) for lung fibrosis. His blood CD4 + T cells count was 255/uL the CD4 + T cells /CD8 + T cells ratio was 0.6, and he had severe lymphocytopenia. He also suffered from pneumonia and severe fungal infection in his mouth and skin. Cessation of the treatment with prednisone and azathioprine lead to the reversal of the damage in his immune system. He fully recovered from pneumonia and the fungal infection after a short course of antibiotics and the use of antifungal lotion. Twenty two days after the last dose of prednisone, his CD4 + T cells count was back to normal at 657 cells/uL (Al-Bayati, 1999)