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In mice with diet-induced diabetes -- the equivalent of type 2 diabetes in humans -- a single injection of the protein FGF1 is enough to restore blood sugar levels to a healthy range for more than two days. The discovery by Salk scientists, published today in the journal Nature, could lead to a new generation of safer, more effective diabetes drugs.
The team found that sustained treatment with the protein doesn't merely keep blood sugar under control, but also reverses insulin insensitivity, the underlying physiological cause of diabetes. Equally exciting, the newly developed treatment doesn't result in side effects common to most current diabetes treatments.
"Controlling glucose is a dominant problem in our society," says Ronald M. Evans, director of Salk's Gene Expression Laboratory and corresponding author of the paper. "And FGF1 offers a new method to control glucose in a powerful and unexpected way." Type 2 diabetes, which can be brought on by excess weight and inactivity, has skyrocketed over the past few decades in the United States and around the world. Almost 30 million Americans are estimated to have the disease, where glucose builds up in the bloodstream because not enough sugar-carting insulin is produced or because cells have become insulin-resistant, ignoring signals to absorb sugar. As a chronic disease, diabetes can cause serious health problems and has no specific cure. Rather it is managed -- with varying levels of success -- through a combination of diet, exercise and pharmaceuticals.
The protein encoded by this gene is a member of the fibroblast growth factor (FGF) family. FGF family members possess broad mitogenic and cell survival activities, and are involved in a variety of biological processes, including embryonic development, cell growth, morphogenesis, tissue repair, tumor growth and invasion. This protein functions as a modifier of endothelial cell migration and proliferation, as well as an angiogenic factor. It acts as a mitogen for a variety of mesoderm- and neuroectoderm-derived cells in vitro, thus is thought to be involved in organogenesis. Three alternatively spliced variants encoding different isoforms have been described. In mice with diet-induced diabetes, the equivalent of type 2 diabetes in humans, a single injection of the protein FGF1 is enough to restore blood sugar levels to a healthy range for > 2 days
How Cinnamon Can Benefit Diabetics
Below are five known ways cinnamon can be helpful to your metabolism:
1. Cinnamon can increase your glucose metabolism about 20-fold, which significantly improves blood sugar regulation. (4)
2. Cinnamon has been found to have "insulin-like effects" due to a bioactive compound, qualifying it as a candidate for an insulin substitute.
3. Cinnamon slows the emptying of your stomach to reduce sharp rises in blood sugar following meals, and improves the effectiveness, or sensitivity, of insulin.
4. Cinnamon actually enhances your antioxidant defenses. A study published in 2009 stated, "Polyphenols from cinnamon could be of special interest in people who are overweight with impaired fasting glucose since they might act as both insulin sensitizers and antioxidants." (5)
5. A bioflavonoid found in cinnamon called proanthocyanidin may alter the insulin-signaling activity in your fat cells.
Other health benefits of cinnamon include:
• Supporting digestive function
• Relieving congestion
• Relieving pain and stiffness of muscles and joints
• Reducing inflammation and symptoms of arthritis
• Helping to prevent urinary tract infections, tooth decay and gum disease
• Relieving menstrual discomfort
• Stimulating circulation with blood-thinning compounds
originally posted by: an0nThinker
a reply to: Char-Lee
A good diet will also reverse or reduce diabetes significantly.
originally posted by: Char-Lee
a reply to: rickymouse
I think the reason for diabetes is that we cook everything to well, worrying about the bad bacteria in the foods now.
Add soda, sweetened coffee donuts....and such.