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"After living and working in China for over 10 years and speaking fluent Chinese, you get to know a society pretty well... and let me tell you this - if you're applauding or admiring the political leadership of China, you're all deluded beyond belief."
That is how "laowhy86" begins this succinct video exploring the 'facts' - not conspiracies - behind the source of the coronavirus that is ravaging the earth.
"China doesn't operate like 'your' country," he warns, "the Chinese government is a face- and greed-driven government that relies on lies and bullying to maintain leadership."
Furthermore, he notes, the Chinese government layers are "broken and fragile" and so it didn't surprise him when he was able to follow breadcrumbs - as begun by our inquisition about the roles that certain individuals played in Wuhan - to discover the "very suspicious" fact that the Wuhan Bio lab had a job opening from November 18, 2019, asking for scientists to come and research the relationship between the coronavirus and bats.
However, after ZeroHedge was permanently suspended from Twitter for daring to suggest anything but the official narrative handed down, laowhy86 notes that another job opening appeared on December 24th (remember this is before any news broke of the virus publicly), which basically says 'we've discovered a new and terrible virus and would like to recruit people to come deal with it'...
Residents in Wuhan, the origin of the coronavirus outbreak, have told reporters that “nobody believes the official numbers” being touted by the communist Chinese government, and that the general consensus is it could be 100 times worse than the world is being told.
While Whuan is reopening its transport systems and businesses, residents are adamant that they have not been informed of the true scale of the situation, according to Spanish newswire service EFE.
“There are suspicions that many people died in their homes without being diagnosed and, at first, there were no kits to do the test,” one resident said, adding that “Many people would die, officially, of a cold or some other disease. But there are stories going around of citizens who were made to sign death certificates of relatives without any other explanation.”
That is how "laowhy86" begins this succinct video exploring the 'facts' - not conspiracies - behind the source of the coronavirus that is ravaging the earth.
The scientists found that the RBD portion of the SARS-CoV-2 spike proteins had evolved to effectively target a molecular feature on the outside of human cells called ACE2, a receptor involved in regulating blood pressure. The SARS-CoV-2 spike protein was so effective at binding the human cells, in fact, that the scientists concluded it was the result of natural selection and not the product of genetic engineering.
This evidence for natural evolution was supported by data on SARS-CoV-2's backbone -- its overall molecular structure. If someone were seeking to engineer a new coronavirus as a pathogen, they would have constructed it from the backbone of a virus known to cause illness. But the scientists found that the SARS-CoV-2 backbone differed substantially from those of already known coronaviruses and mostly resembled related viruses found in bats and pangolins.
"These two features of the virus, the mutations in the RBD portion of the spike protein and its distinct backbone, rules out laboratory manipulation as a potential origin for SARS-CoV-2" said Andersen.
www.sciencedaily.com...
RESEARCH ARTICLE
MICROBIOLOGY
On the origin and continuing evolution of SARS-CoV-2
ABSTRACT
The SARS-CoV-2 epidemic started in late December 2019 in Wuhan, China, and has since
impacted a large portion of China and raised major global concern. Herein, we investigated
the extent of molecular divergence between SARS-CoV-2 and other related coronaviruses.
Although we found only 4% variability in genomic nucleotides between SARS-CoV-2 and a bat SARS-related coronavirus (SARSr-CoV; RaTG13), the difference at neutral sites was 17%, suggesting the divergence between the two viruses is much larger than previously estimated.
Our results suggest that the development of new variations in functional sites in the receptor-binding domain (RBD) of the spike seen in SARS-CoV-2 and viruses from pangolin SARSr-CoVs are likely caused by mutations and natural selection besides recombination.
Population genetic analyses of 103 SARS-CoV-2 genomes indicated that these viruses evolved into two major types (designated L and S), that are well defined by two different SNPs that show nearly complete linkage across the viral strains sequenced to date.
Although the L type (~70%) is more prevalent than the S type (~30%), the S type was found to be the ancestral version. Whereas the L type was more prevalent in the early stages of the outbreak in Wuhan, the frequency of the L type decreased after early January 2020.
Human intervention may have placed more severe selective pressure on the L type, which might be
more aggressive and spread more quickly. On the other hand, the S type, which is evolutionarily older and less aggressive, might have increased in relative frequency due to relatively weaker selective pressure.
These findings strongly support an urgent need for
further immediate, comprehensive studies that combine genomic data, epidemiological data,
and chart records of the clinical symptoms of patients with coronavirus disease 2019
(COVID-19).
Keywords: SARS-CoV-2, virus, molecular evolution, population genetics
Coronavirus strains are maintained in quasi-species pools circulating in bat populations. (a,b) Traditional SARS-CoV emergence theories posit that host-range mutants (red circle) represent random and rare occurrences that permit infection of alternative hosts.
The secondary-host paradigm (a) argues that a nonhuman host is infected by a bat progenitor virus and, through adaptation, facilitates transmission to humans; subsequent replication in humans leads to the epidemic viral strain.
The direct paradigm (b) suggests that transmission occurs between bats and humans without the requirement of an intermediate host; selection then occurs in the human population with closely related viruses replicating in a secondary host, permitting continued viral persistence and adaptation in both.
(c) The data from chimeric SARS-like viruses argue that the quasi-species pools maintain multiple viruses capable of infecting human cells without the need for mutations (red circles). Although adaptations in secondary or human hosts may be required for epidemic emergence, if SHC014 spike–containing viruses recombined with virulent CoV backbones (circles with green outlines), then epidemic disease may be the result in humans. Existing data support elements of all three paradigms.
originally posted by: choomsuba
The answer would be governments that don’t have forced slave labor and cult of personality Dear leaders that are in charge for their entire life until they die.
reply to: McGinty
originally posted by: HelloboysImbackguy
a reply to: choomsuba
Lets consider the facts. Im weird and read the reports and studies too. Lets try to use reason and facts.
This Virus is 8 to 10 times slower mutating than an influenza virus. Its very genetically stable.
That means its traits have been reinforced and are part of the unchanging code it replicates.
originally posted by: gortex
a reply to: choomsuba
“Those guys”are paid shills for the Chinese government
Of course they are dear , of course they are.
originally posted by: HelloboysImbackguy
a reply to: 727Sky
Welp, thats obvious.
When do we start charging China for our stimulus checks and loss income? I heard a US lawyer sued the CCP for 2 trillion.
If they dont pay, do we bomb them? No. I say we just nullify the debt they own. Call it even.
Really I want to kick CCP ass and erase that lab off the map.
Blood for blood.
Clinical features in humans
Mild form of RVF in humans
The following are clinical features of the mild form of RVF in humans:
The incubation period (the interval from infection to onset of symptoms) for RVF varies from 2 to 6 days.
Those infected either experience no detectable symptoms or develop a mild form of the disease characterized by a feverish syndrome with sudden onset of flu-like fever, muscle pain, joint pain and headache. Some patients develop neck stiffness, sensitivity to light, loss of appetite and vomiting; in these patients the disease, in its early stages, may be mistaken for meningitis.
The symptoms of RVF usually last from 4 to 7 days, after which time the immune response becomes detectable with the appearance of antibodies and the virus disappears from the blood