It looks like you're using an Ad Blocker.
Please white-list or disable AboveTopSecret.com in your ad-blocking tool.
Thank you.
Some features of ATS will be disabled while you continue to use an ad-blocker.
originally posted by: frogs453
That's the problem with fear mongering. Just jumping to one conclusion without considering all of the possibilities. Why is the thread not considering all of the possibilities?
originally posted by: frogs453
originally posted by: putnam6
originally posted by: frogs453
I do know of a beautiful 28 year old mom of two who died on her front lawn while walking to her car of a stroke, and my uncle had his first stroke at 30. However, this was well before 2020. Both appeared very fit and healthy at the time. It does occur.
FWIW it can be both, yes young people have died before, but the question is how many are dying? there are a few reputable people saying we are now getting excess deaths above the normal "young people dying"
Possibly. However, why does everyone jump to vaccines, when it was discovered before the vaccinations were available that even an asymptomatic case of covid can cause heart attacks and stokes months after the covid case? So, the thing is, much more information is needed before you can assume it's the vaccination. That's the problem with fear mongering. Just jumping to one conclusion without considering all of the possibilities. Why is the thread not considering all of the possibilities?
originally posted by: frogs453
a reply to: MaxxAction
Here is some information.
Link
Link
Link
And more. Link
There are many more if you wish.
Post COVID-19 infection was not associated with either myocarditis (aHR 1.08; 95% CI 0.45 to 2.56) or pericarditis (aHR 0.53; 95% CI 0.25 to 1.13). We did not observe an increased incidence of neither pericarditis nor myocarditis in adult patients recovering from COVID-19 infection
The study shows that the immunological response elicited by the mRNA vaccine was very similar in those who developed post-vaccination myocarditis and others. In other words, myocarditis could not be associated with abnormal autoantibodies, viral infections other than SARS-CoV-2, or excessive production of antibodies elicited by the mRNA vaccine.
In vaccinated patients, infection with the virus was not likely to be a cause or contributing factor for myocarditis since anti-Nucleoprotein IgG was not found in these patients.
In contrast to controls, the finding of high levels of unbound full-length spike protein in myocarditis patients may point to the mechanism by which this condition arises. Similarly, MIS-C patients had circulating SARS-CoV-2 antigens.
The spike protein appears to evade immune antibodies found at normal levels in these patients, with adequate functional and neutralization capacity. The spike may damage the cardiac pericytes or endothelium, perhaps by reducing the expression of the angiotensin-converting enzyme 2 (ACE2), reducing nitric oxide production in the endothelium, or activating inflammation via integrins, causing the endothelium to become abnormally permeable