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originally posted by: Kenzo
I forget to add, could be 5G issue
Can 5G exposure alter the structure and function of hemoglobin, causing coronavirus patients to die from oxygen deprivation?
But im sure many here will declare 5G a non issue again .
The past 48 hours or so have seen a huge revelation: COVID-19 causes prolonged and progressive hypoxia (starving your body of oxygen) by binding to the heme groups in hemoglobin in your red blood cells
. People are simply desaturating (losing o2 in their blood), and that’s what eventually leads to organ failures that kill them, not any form of ARDS or pneumonia. All the damage to the lungs you see in CT scans are from the release of oxidative iron from the hemes, this overwhelms the natural defenses against pulmonary oxidative stress and causes that nice, always-bilateral ground glass opacity in the lungs.
Patients returning for re-hospitalization days or weeks after recovery suffering from apparent delayed post-hypoxic leukoencephalopathy strengthen the notion COVID-19 patients are suffering from hypoxia despite no signs of respiratory ‘tire out’ or fatigue.
But im sure many here will declare 5G a non issue again .
Humanity is not a virus. Covid-19 is an attack on humanity. Everyone in America can test positive for Covid-19 assuming they have an immune system. What they're calling a test is not a test - its a scarlet letter. The coronavirus is the body's exosomes. These are secreted by the cells under numerous conditions. Duke-trained Doctor Andrew Kaufman explains it all in a concise 30-minute presentation. After, you hear laymen like myself ask questions. Please share, steal, reupload, monetize, condense, and spread as you see fit. You are the land's immune system. This video is everything you need to spread the word. Call out the Minutemen who will still listen. Tell them we need them now more than ever.
A formalin-inactivated RVFV vaccine, TSI-GSD-200, has been developed; however, it is not licensed and not commercially available (Pittman et al., 1999). TSI-GSD-200 is only provided to veterinarians working in endemic areas, high containment laboratory workers and others at high risk for contracting RVFV (Pittman et al., 1999). Unfortunately, th
Unfortunately, this vaccine is (i) expensive, (ii) difficult to produce, (iii) in short supply, (iv) requires larger dose relative to an attenuated vaccine and three initial inoculations followed by a 6-month booster (v) and requires continued annual boosters to maintain protective immunity (Frank-Peterside, 2000, Kark et al., 1982, Kark et al., 1985, Niklasson et al., 1985).
The use of virus-like particles (VLPs) is a promising approach for the development of a safe and efficient RVFV vaccine.
Expression of structural proteins of many non-enveloped and enveloped viruses leads to the formation of VLPs (Garcea and Gissmann, 2004, Grgacic and Anderson, 2006a, Grgacic and Anderson, 2006b, Noad and Roy, 2003).
Such VLPs frequently exhibit a morphology very similar to that of wild-type (wt) viruses (Johnson and Chiu, 2000).