I have been interested in the RH negative blood antigen for a long time but recently discovered that there may be a connection between the RH- and
certain autoimmune diseases. This may add to the "Extraterrestrial Threat to those with negative blood types?" thread
and a new thread here "possible threat to some people and the conspiracy to hide it all until
it's too late" www.abovetopsecret.com...
The name of this thread is "Possible link" because there seems to be a link with RH- blood and ankylosing spondylosis (vertebrae become fused
together) but I can not verify it completely. I hope that someone can fill in some of the blank spots.
First I would like to recap the various blood type groupings. There are four basic blood types: O, A, B and AB. Your type will come from both of your
parents and types can be determined through a blood test. Type O is recessive so to have this type both parents must have type O and it can be either
dominant or recessive in the parent’s blood type. The blood type that shows in the standard blood test will show the dominant type only. To
determine your recessive type you must include types from both parents if their types are different.
More information here:
The RH factor is also determined through a standard blood test and indicates the presence (RH+) or absence (RH-) of an antigen that is also present in
Rhesus monkeys. RH- is only found in humans.
Parent A Parent B Child
RH+ RH+ RH+
RH+ RH- RH+ (with recessive RH- or RH+/-)
RH- RH- RH-
I know that some people like to say that the RH- indicates a reptilian influence but if this were true I would think that there would be a higher
incidence of eczema, psoriasis or similar type of skin condition producing “scaly” skin. I have not been able to find this connection. It may be
that doctors have not made the connection or a case of doctor-patient confidence where the data is not available for evaluation. It also might not
matter to the doctor because the treatment would be the same regardless of blood type.
The most widely known autoimmune response involving RH- is RhD Hemolytic Disease where an RH- mother is carrying an RH+ child. If their blood mixes it
triggers an autoimmune reaction in the mother to attack and reject the fetus. It rarely if ever happens in the first pregnacy but can happen on
subsequent pregnacies. There is no similiar reaction if an RH+ mother carries an RH- child.
Rh disease (also known as Rh (D) disease, Rhesus incompatibility, Rhesus disease, RhD Hemolytic Disease of the Newborn, Rhesus D Hemolytic Disease of
the Newborn or RhD HDN) is one of the causes of hemolytic disease of the newborn (HDN). The disease ranges from mild to severe, and typically occurs
only in some second or subsequent pregnancies of Rh negative women where the fetus' father is Rh positive,leading to a Rh+ pregnancy. During birth,
the mother may be exposed to the infant's blood, and this causes the development of antibodies, which may affect the health of subsequent Rh+
pregnancies. In mild cases, the fetus may have mild anaemia with reticulocytosis. In moderate or severe cases the fetus may have a more marked anaemia
and erythroblastosis (erythroblastosis fetalis). When the disease is very severe it may cause HDN, hydrops fetalis, or stillbirth.
Rh disease is generally preventable by treating the mother during pregnancy or soon after delivery with an intramuscular injection of anti-RhD
immunoglobulin (Rho(D) immune globulin).
With first pregnancies, Rh incompatibility usually does not pose a problem, but with subsequent pregnancies, the risks increase.
What makes Rh incompatibility so concerning is that if it is not detected, your baby can develop severe jaundice. Jaundice, if left untreated, can
cause brain damage. And in turn, brain damage can cause cerebral palsy.
The HLA-B27 marker is known to be associated with rheumatoid arthritis and ankylosing spondylosis. There may be a link between this and RH- blood but
I have been unable to verify this to my satisfaction. All of the sources I have been able to find are blog type sites and quote the same article from
Randall Johnson at the Baylor College of Houston University. The article states the HLA-B27 marker is only found in people with RH- blood but they
offer no substaining sources or links. I have been able to verify that there is someone named Randall Johnson at Baylor and he is involved in this
type of research but found nothing linking him to the article so I can not say for sure if this is correct and am hoping someone can help out. I did
find a couple of blogs where they asked people with ankylosing spondylosis if they had the HLA-B27 marker and if they were RH- or not. Most were RH-
but there were some RH+ as well. They did not however say if there was a possible recessive RH-.
All of the sites with this connection have postings like this with no supporting documentation:
According to Randall Johnson at the Baylor College of Medicine in Houston, "Only 7% of the US population tests positive for the HLA-B27 gene; this
gene, found only in persons with Rh-Negative blood, can trigger the immune system to operate overtime at WARP SPEED in times of medical emergency."
Information about HLA-B27 and diseases:
The HLA-B27 Genetic Marker is said to have protective properties that guard against the progression of HIV. It is said, that people with this gene do
not have the right proteins for the HIV virus to bind with. The HLA - B27 Marker is most often found in people with O- Blood.
HLA-B27 is an inherited gene marker that is associated with a number of related rheumatic diseases. They share in common, certain features like
spinal and peripheral arthritis, skin and GI disorders, anterior chamber eye disease, psoriasis like skin lesions, as well as inflammation and joint
pain. This gene is found with highest prevalence in patients with ankylosing spondylosis, reactive arthritis, and patients with the combination of
peripheral arthritis and either psoriasis or inflammatory bowel disease.
This is a site with a lot of information about RH- but unfortunately most requires paid membership to view. Noted quote was taken from public
A major genetic link was discovered back in the 1960's between the gene HLA-B27 and the spondyloarthropathies, a group of diseases affecting the spine
and other joints.
•90 percent of people with ankylosing spondylitis have the HLA-B27 gene.
•7 percent of the general population have the HLA-B27 gene.
Scientists are working hard to find the other genes involved in the spondyloarthropathies and other rheumatic diseases.
During the decade following the discovery of HLA-B27, scientists found an association between carriers of the HLA-DR genes and increased risk of
rheumatoid arthritis. It is believed that HLA-DR contributes to autoimmune disease, conditions caused by the immune system fighting the body it is
supposed to protect. Other genes also are suspected of being major factors in the evolution of rheumatoid arthritis.
Information about ankylosing spondylosis:
edit on 7/18/2011 by LuFri because: (no reason given)