The symptoms of GWS are generally Sleep Problems, Mood Swings, Short-term Memory Loss, Headaches, Sensitivity to bright light, Blurred vision/Loss of
focus, Chronic Fatigue, Rashes, Aching Joints, Abdominal Pain, Diarrhea, Respiratory problems, Cardiovascular problems.
Cirrosis (spelling) and Necrosis of the liver have also been commonly identified with Gulf War Syndrome.
Other phenomenon associated with GWS in some studies are
1. Human Endogenous Retro-Viruses (HERVs) and failure to develop antibodies when vaccinated (signs of immune failure),
2. fungal- or protozoa-like structures, decreased protein, increased pH resulting in enhanced polyamine synthesis and ammonia in semen- resulting in a
burning sensation for the victims wife during intercourse.
3. Peripheral neuropathies.
This is also found at www.gulfweb.org...
under the heading "private medical research".
The research which I reference next is published here: www.swmed.edu...
and was debated in the Journal of the American
Medical Association, which you can read here: www.gulfweb.org...
UT Southwestern researchers under Dr. Haley conducted a study of 249 members of a US Navy Reserve Construction Battalion and found that 1/4 of them
suffered groupings of symptoms. Three groupings of neurological symptoms were discovered. This represents some of the pioneering research in the
challenge to the government's ridiculous assertion that GWS is a somatiform related to Post Traumatic Stress Disorder, and establishes the
neurological basis of GWS. THESE FINDINGS SHOULD NOT DISCOURAGE INTEREST IN NON-NEUROLOGICAL EXPLANATIONS, as they are only a beginning, and do not
perfectly define all cases of GWS, nor have they yet yielded anything of use in diagnosis and treatment to my knowledge. This information was heavily
discussed in the Journal of the American Medical Association, and finds its chief opposition from doctors employed at Bethesda, who have a large stake
in the lie that GWS is stress-related.
The three classes of neurological symptoms identified in 1/4 of the group of 249 Naval Reservists by Dr. Haley:
The first, titled "Impaired Cognition" manifested in the form of diarhea, distractability, difficulty remembering, depression, insomnia, daytime
sleepiness, slurred speach, confusion, and migraines. The common risk factors among affected veterans were use of flea-collars intended for pets,
employment in security roles, and youth. These symptoms mirror the DoD definition of GWS.
The second, titled "Confusion Ataxia" manifested as diarhea, disorientation, dizziness/vertigo, difficulty reasoning, and sexual impotence. Common
risk factors were experience of side-effects from Pyridostigmine Bromide pills, having been under suspected chemical attack (fired upon by scud
missiles), especially at Khafji on Jan. 20, 1991, and older age.
The third, titled "Arthro-myo-neuropathy" manifested as diarhea, joint/muscle pain, weakness, or fatigue as well as numbness or tingling in the
hands and feet. Common risk factors were older age, side effects of Pyridostigmine Bromide pills, and use of 75% DEET insect repellant.
These veterans were not in close proximity to the accidental sarin release caused by the 37th engineer battalion at Khamisiyah at any point during the
war. Stress has been ruled out as a causative factor both by this study and by the British Ministry of Defence.
From this study we find 2 distinct neurological disorders tied to the combination of organo-phosphates and PB (in one case by chemical attack and PB
resulting in confusion ataxia, and in the other case by DEET and PB resulting in Artho-myoneuropathy.)
I say 2 because it seems reasonable that the first set, "impaired cognition" is a milder form of "confusion ataxia" as these veterans are noted to
be younger and to have selected an alternative to Organophosphate insect repellants. The result here is the identification of two neurological
explanations of gulf war syndrome.
It bears emphasis that this study was aimed at the identification of a neurological explanation, and does not address the presence of fungus or the
immune failure reported by other research. It is necessary to search either for additional effects of the above-named causes or for additional causes.
Also, it is important to discover the link between gastric problems and neurological disorders, as well as why in some cases these problems seem to
have been passed on to family members.
The next piece of information I found clarified this for me, and showed me how a common factor could have caused neurological damage, respiratory
damage, gastric damage, and the entire range of symptoms identified by gulf war veterans.
The following is the account of an incident that happened to a Naval Reserve consturction unit- I can not confirm that it is the same unit which Dr.
Haley tested, but it is possible. When you read this, remember that it happened on 19 or 20 January (depending on which source you read). Being in a
certain location on 20 January was named as a risk factor for Confusion Ataxia, the second and most severe syndrome identified by Dr. Haley! The quote
is taken from www.mold-help.org...
At about 3:00 AM on the morning of January 19, a brilliant flash of red light appeared in the cloud-covered sky over the port of Al Jubayl. The flash
was followed by a loud double-explosion and a shockwave powerful enough to knock over tents at Camp 13 and awaken the sleeping troops. According to
one eyewitness, "The concussion was so strong that it knocked me to my knees." 
Almost immediately, the general-quarters alarm began to sound. Thomas Harper, communications chief for the Air Detachment, heard a warning message
come over the camp radio net: "Alpha 6 Bravo (the unitís call sign), we have a confirmed chemical agent." The radio then broadcast a message to all
stations ordering the troops to don their gas masks and chemical-protective suits and proceed to bunkers. There was also a call for chemical
decontamination teams. 
Near-panic broke out as the Seabees struggled to pull on their masks and rubberized suits. As the troops emerged from their tents and ran to the
bunkers, they smelled a sharp, acrid odor and saw a dense yellowish mist floating over the camp. Many individuals did not mask in time or failed to
achieve a good seal. They began to choke, and a few had profuse nasal secretions that fouled (clogged) their masks. Fred Willoughby felt his mouth,
lips, and face go numb, a sensation similar to Novocain at a dentistís office. Roy Morrow and Nick Roberts experienced a burning sensation on their
exposed skin and a strange metallic taste, "like sucking on a penny." 
When the sun rose later that morning, Nick Roberts observed a thin coating of yellow powder on tents and vehicles. Other Seabees noticed that an area
near the entrance to the commercial port of Jubayl had been cordoned off with yellow hazard tape, and that a fenced-in herd of animals nearby had
died. Later that day, the battalionís chemical officers collected each soldierís chemical-protective suit and replaced it with a new one, the only
time such a gear exchange took place during the war. Because of a shortage of equipment, routine replacement of chemical suits was extremely rare
during the Gulf War, if it occurred at all.
Within a few days after the mysterious double-explosion, those Seabees who had experienced burning skin, facial numbness, and choking began to suffer
from flu-like symptoms, including fever, sweating, diarrhea, and muscle cramps and spasms. Areas of skin that had been exposed during the incident
broke out in rashes, welts, and small blisters, which eventually burst and turned into ulcerating sores that scabbed over and healed but later
recurred. The Seabeesí joints also began to ache, and the pain became progressively worse. Several developed painfully swollen lymph nodes.
This website deals with the effects of Mycotoxins and the molds with which they are associated.
Mycotoxins are diverse secondary metabolites produced by fungi growing on a variety of foodstuffs consumed by both animals and humans (Table
2) (76). Clinical toxicological syndromes caused by ingestion of large amounts of mycotoxins have been well characterized in animals and range from
acute mortality to slow growth and reduced reproductive efficiency. The effects on humans are much less well characterized
Reduced reproductive efficiency... sexual impotence was part of the symptoms included in Dr. Haley's syndromes.
Mycotoxins are probably responsible for a range of acute and chronic effects that cannot be attributed to fungal growth within the host (301)
or allergic reactions to foreign proteins (370).
Thats why it's hard to identify.
The most notorious and best described of the mycotoxins are the aflatoxins... The aflatoxins are toxic, immunosuppressive, mutogenic,
teratogenic, and carcinogenic, and their main target is the liver. Most have been classified as type 1 carcinogens
This is where the abdonimal pain and cases of liver cancer and necrosis are coming from. This also explains the ineffectiveness of vaccines and the
problem with retro-virusses experienced by veterans, as well as the birth defects of their children.
In the Ukraine in the early 1930s, a unique disease of horses was recognized that was characterized by lip edema, stomatitis, oral necrosis...
neurologic compromise (irritability, gait disturbance, and blindness), superinfections, and finally death (133). There was also a rare "atypical"
form, which was primarily neurological and highly fatal, with areflexia (loss of sensorimotor reflexes), hyperesthesia (hypersensitivity to pain),
hyperirritability, blindness, and stupor.
Mycotoxins are known to cause mood swings, confusion, a disturbance of motor skills- signs of GWS.
Sites of action include initiation of protein synthesis (scirpentriol, 15-acetoxyscirpendiol, DAS, verucarin A, and T-2 toxin) and elongation
or termination (trichodermin, trichodermol, crotocol, trichothecolone, trichothecin, and verrucarol) (187, 258). Because of their potency in affecting
protein synthesis, they may cause a predilection to other diseases, masking the underlying toxicosis
Remember the earlier source which mentioned increased protien and pH in semen, and the occurence of retro-viruses? The above quote makes sense in that
light, doesn't it?
While S. chartarum produces several very toxic macrocyclic trichothecenes (32, 150-152, 183, 188), the levels at which these toxins are
produced in laboratory cultures have never appeared sufficient to cause such profound toxic effects as have been observed in animals (188). Levels are
also low in environmental samples. Chemical analysis of such samples is difficult due to intrinsic compound properties and secondary metabolite
production; despite much work, most potential products are uncharacterized
Another indication of why it's hard to identify and treat. You could easily argue that it's not harmful, yet we know that it's harming animals. The
same way the VA argues that GWS isn't from a harmful physical source, even though it's harming veterans.
The most famous purported case of mass human trichothecene toxicity was in fact due to Fusarium. The illness was initially dubbed "septic sore
throat" and subsequently called alimentary toxic aleukia (ATA) (7, 193-195, 252, 253, 274, 369, 380, 408, 429). It occurred in Russia in the early
20th century, most notably prior to and during World War II, and was characterized by several stages. Initially, there was oral mucosal ulceration and
gastroenteritis. Subsequently, there was pancytopenia accompanied by fatigue, vertigo, and hypotension. The illness had a substantial mortality rate,
at least in part due to opportunistic bacterial infections developing in the later stages of the disease (53, 69, 370). One-third of family members
who ate contaminated grain became ill, and one-third of those died; this was responsible for thousands of deaths. Where nutrition was good, morbidity
and mortality were much lower
Gastric problems, fatigue, vertigo, hypotension, opportunistic diseases... sounds vaguely familiar.
Verruculogens and penitrem A may be "tremorgenic mycotoxins," responsible for tremors, ataxia, weakness, and convulsions in animals
What do you know... not just generic neurological problems, but a form of ataxia, just like Haley identified in GWS victims.
This website deals with the treatment of Mycotoxicosis.
One thing I note is that this website refers the the symptoms as Chronic Fatigue Syndrome (CFIDS). A little reading into my sources on GWS will reveal
that CFIDS is often mentioned right along side, BECAUSE THE SYMPTOMS ARE THE SAME!
Wrapping up for the moment, I appologize for the length of this post. I did my best to consolidate 3 days worth of study into a single post without
ommitting too much of many citations of evidence. I may edit this for neatness later.