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He is coauthor of a #1 Amazon.com bestseller, Born A Healer, and coauthor with Dr. Nisha Manek of the Mayo Clinic of a chapter on qigong in the recently-released Textbook of Complementary and Alternative Medicine, 2nd Edition.
Medical Research and Spring Forest Qigong Two new research studies, one by doctors at the Mayo Clinic, have found Spring Forest Qigong techniques effective in relieving Chronic Pain. A brief synopsis of each study is below. The studies are soon to be published. We will provide complete information as soon as it becomes available. External Qigong for Chronic Pain, Results from a Randomized Controlled Clinical Study Jamia Hill, RN, DC; Anne Vincent, MD, Stephen Cha, PhD., Nisha Manek, MD, MRCP Mayo Clinic, Rochester, MN Objective: Chronic pain is highly prevalent in the general population. Adequate clinical management of chronic pain is an ongoing challenge and a purely pharmaceutical approach has proven inadequate. We investigated the efficacy of external Qigong [Spring Forest Qigong technique] as an adjunctive treatment for chronic pain. Conclusions: Subjects with chronic pain who received external Qigong experienced reduction in pain intensity following each Qigong treatment. This is especially impressive given the long duration of pain in the majority of subjects. Utilizing Spring Forest Qigong as a Self-Directed Treatment for Chronic Pain & Emotional Distress Jane F. Coleman, R.N., PhD Professor Emerita, Gustavus Adolphus College, St. Peter, MN Study Findings: There was a significant decrease in the perception of physical pain and emotional distress for the majority of participants during the study timeframe. Also, symptom variables (sleep, concentration, decision-making, appetite, loss of interest) improved in the majority of subjects. Conclusion: Both the active exercise and meditative aspects of Spring Forest Qigong proved to be effective self-care modalities for persons with perceived chronic physical pain and/or emotional distress. Subjects demonstrated significant improvement both anecdotally and statistically during the study period.
Int J Biochem Cell Biol. 2006;38(12):2102-13. Epub 2006 Jun 27. External Qi of Yan Xin Qigong differentially regulates the Akt and extracellular signal-regulated kinase pathways and is cytotoxic to cancer cells but not to normal cells. Yan X, Shen H, Jiang H, Zhang C, Hu D, Wang J, Wu X. Institute of Chongqing Traditional Chinese Medicine, Chongqing, PR China. email@example.com Long-term clinical observations and ongoing studies have shown significant antitumor effect of external Qi of Yan Xin Qigong which originated from traditional Chinese medicine. In order to understand the molecular and cellular mechanisms underlying the antitumor effect of external Qi of Yan Xin Qigong, we have examined its cytotoxic effect on BxPC3 pancreatic cancer cells and its effect on the Akt and extracellular signal-regulated kinase pathways. We found that external Qi of Yan Xin Qigong dramatically inhibited basal phosphorylation levels of Akt and extracellular signal-regulated kinases, epidermal growth factor-mediated phosphorylation of extracellular signal-regulated kinases, and phosphatidylinositol 3-kinase activity. External Qi of Yan Xin Qigong also inhibited constitutive and inducible activities of nuclear factor-kappa B, a target of the Akt and epidermal growth factor receptor pathways. Furthermore, a single 5min exposure of BxPC3 cells to external Qi of Yan Xin Qigong induced apoptosis, accompanied by a dramatic increase of the sub-G1 cell population, DNA fragmentation, and cleavage of caspases 3, 8 and 9, and poly(ADP-ribose) polymerase. Prolonged treatment with external Qi of Yan Xin Qigong caused rapid lysis of BxPC3 cells. In contrast, treatment of fibroblasts with external Qi of Yan Xin Qigong induced transient activation of extracellular signal-regulated kinases and Akt, and caused no cytotoxic effect. These findings suggest that external Qi of Yan Xin Qigong may differentially regulate these survival pathways in cancer versus normal cells and exert cytotoxic effects preferentially on cancer cells, and that it could potentially be a valuable approach for therapy of pancreatic carcinomas.