Deadly Diabetes Deception

There are two types of diabetes...Type 1 and Type 2

Type 1 Diabetes has been known for most of human history.
Type 1 currently accounts for only about 5% of all diabetes.
Essentially it is the failure of the Pancreas to produce insulin.
It can be cause by an organism destroying the cells that produce insulin.
It can be caused by an auto-immune complex.
It can be caused by trauma to the pancreas.

Type 2 Diabetes has only been known since the 1930’s *
Type 2 currently accounts for about 95% of all diabetes.
It was a rare disease prior to the 1930's...it is now an epidemic....why?
It is also known as…
Insulin resistance diabetes
Syndrome-x
Adult onset diabetes

Essentially it is the failure of cells to absorb and process sugars.
It is also associated with other nutrient deficiency disorders.
However, Type 2 diabetes is not caused by sugars…it is caused by fats.

Prior to the 1930’s the predominant edible fats were...
butter
flax/linseed oil
coconut oil
fish oils…
...then something changed.

Early in the 1900’s science had been produced, discovered and/or engineered new fats.
These new oils were essentially of vegetable origin.
They were often obtained using heat extraction methods that damaged the nutritional value.
These were mostly short to medium chain fatty-acids.
There was no market for them while the ‘traditional’ oils were being used.
They began to be used during WW2 when the traditional oils were in short supply.

What scientists now know is that fats are used by the body to construct cell membranes.
The traditional food oils produce supple slippery cell membranes.
These normal cell membranes allow for the easy transport of sugars and nutrients.

The new engineered oils produce stiff sticky cell membranes.
These restrict the normal absorption of sugars and nutrients.

The result is elevated blood sugar levels and nutrient deficiency syndromes.

Type 2 diabetes is essentially a feedback crisis…
The cell starves for sugar and signals for more to be released.
Blood sugar levels rise…
The pancreas releases more insulin to compensate.
The cell continues to demand more sugar.
The feedback loop gradually worsens.

Solution:
The process can be reversed…
If all engineered oils (margarines, vegetable oils, heat extract oils) are removed from the diet and are replaced with the traditional oils (pure butter, flax/linseed oil, coconut oil, fish oils) the normal sugar regulation system will return as cells are replaced during normal cell turnover.

The time it takes for the syndrome to reverse will depend on how long the condition has existed and the volume of cells affected. This can take as little as a few months to over twelve months.

References:
* Insulin-stimulated glucose uptake involves the transition of glucose transporters to a caveolae-rich fraction within the plasma cell membrane: implications for type II diabetes", Mol. Med. 2(3):367-372 (May 1996) Gustavson, J., et al

reversingdiabetes.files.wordpress.com...

www.healingmatters.com...



[edit on 21-11-2009 by troubleshooter]

reply to post by troubleshooter

Here's an outline of how oils are made. *

Healthy oils...
- selected seeds - organic pesticide controlled.
- dirt and dust removed.
- hulls and husks removed.
- small scale cold pressed in an oxygen and light free environment.
- filtered.
- nitrogen flushed (removes air bubbles).

Results in an oil as fresh as freshly squeezed orange juice.
These oils build healthy soft/slippery cell membranes…
…that absorb nutrients and sugars.


Engineered oils...
- oil seed of any quality (incl chemically sprayed and fertilized)
- dirt and dust removed.
- hulls and husks removed.
- seed crushed and flaked.
- steam pre-heated 110º F-180º F (45º C-85º C)
- high volume expeller press with friction heat 162º F-183º F (72º C-84º C)
- solvent extraction process – pulp with oil using Hexane solvent.
- extraction of oil from pulp.
- then the oil plus solvent (Micella) is steam heated and centrifugally separated.
- Phosphate added and then distilled using Caustic Soda
- Mucilage process
- Refining, neutralizing, degumming.
- water washing process
- passed through a Diatomaceous earth 2-3% filtration.
- bleaching agent added and filtered.
- Deodorizing 450º F-470º F (230º C-245ºC) using steam injection.

Results in Refined cooking oil used in most processed foods.
These oils are [I]non-foods [/I]that build stiff/sticky cell membranes…
…these produce body cells that poorly absorb nutrients and sugars.

Switch your diet to the cold pressed unrefined oils and your type 2 diabetes will reverse...

* Outline summary from a major oil producer.

Over half of all Americans are overweight or obese. If they exercised & put the fork down the weight would come off.Add high fructose sugar & refined foods and you have a bad mix.

What about adipose fat ??

Originally posted by fishing
Over half of all Americans are overweight or obese. If they exercised & put the fork down the weight would come off.Add high fructose sugar & refined foods and you have a bad mix.

That is the common wisdom...
...excercise only increases cell metabolism, so increases absorption of sugars...
...but this does not change the inefficiency of the cell...
...even coffee/caffiene can increase cell metabolism...
...but this does not change the inefficiency of the cell membrane.

I observed a 5 year Type 2 diabetic with sugar levels of 21 before insulin...
...back in the normal range in six months by changing oil consumption alone.

What about adipose fat ??

Insulin does essentially two things...
...transports sugars into cells...
...transports excess serum sugars to the liver to be converted to adipose fat...
...when good oils produce a critical mass of normal soft/slippery cells...
...the cell stops demanding sugar, serum sugars return to normal, no more adipose fat is produced.

You need to do more research.

There are more then two types of diabetes.
and there is more then one cause for type 2 diabetes

there is 4 types of diabetes insipidus
en.wikipedia.org...

then there is Diabetes mellitus that also has a number of types and causes.
Type 1:
Type 2
Gestational diabetes:
latent autoimmune diabetes of adults
en.wikipedia.org...

Many other forms of diabetes mellitus are categorized separately from these. Examples include congenital diabetes due to genetic defects of insulin secretion, cystic fibrosis-related diabetes, steroid diabetes induced by high doses of glucocorticoids, and several forms of monogenic diabetes.

Other types

Most cases of diabetes mellitus fall into the two broad etiologic categories of type 1 or type 2 diabetes. However, many types of diabetes mellitus have more specific known causes, and thus fall into more specific categories. As more research is done into diabetes, many patients who were previously diagnosed as type 1 or type 2 diabetes will have their condition reclassified.

Many doctor just for simplicity tell the diabetics they treat just type 1 or 2.
because most people do not understand the numerous types.

If you are young under 5 in most cases and must take insulin injections. your type 1

If you are older (over 20 in most cases)and in many cases treated with drugs for years before going on insulin they class you as type 2 for convenience.

Only if you ask the right doctor will you get the right answers as to what subtype of diabetic you are.
and for diabetes that would be a Diabetes Endocrinology Specialist.

Most General Practitioners will never take the time. they just treat you to control your blood sugar and tell you a type by just your age at onset.

Type 2 currently accounts for about 95% of all diabetes.
It was a rare disease prior to the 1930's...it is now an epidemic....why?
It is also known as…
Insulin resistance diabetes
Syndrome-x
Adult onset diabetes

Essentially it is the failure of cells to absorb and process sugars.
It is also associated with other nutrient deficiency disorders.
However, Type 2 diabetes is not caused by sugars…it is caused by fats.

What medical training did you have. I have been a EMT for over 30 years and am a diabetic myself.
I will agree that Insulin resistance diabetes,syndrome-x metabolic syndrome type diabetes is not caused by suger,
The cells in your body still absorb sugars but because the Insulin receptor no longer work right and the cells can not process sugars. This causes weak and in some cases painful muscles because without Insulin and sugar in the cells they can not repair themselves. this in its self can make exercise hard to do.

Excess blood sugar is stored in the liver and other vital organs; when the organs are "full," the excess blood sugar is converted to fat.

When the insulin receptor in your body do not work the liver responds as if your organs are full and because the liver does not need insulin receptors to make fat from insulin and sugar that is what it does with excess insulin and sugar the rest of the body can not use.
The muscles other vital organs can use limited amounts of fat as ketones if you do not get enough sugar for energy without insulin. To much ketones can cause problems for diabetics.
Fat gain is a symptom of Insulin resistance not the cause.
You gain fat because your muscles and organs do not use insulin properly.


en.wikipedia.org...

Pathophysiology

It is common for there to be a development of visceral fat, after which the adipocytes (fat cells) of the visceral fat increase plasma levels of TNFα and alter levels of a number of other substances (e.g., adiponectin, resistin, PAI-1). TNFα has been shown not only to cause the production of inflammatory cytokines but possibly to trigger cell signaling by interaction with a TNFα receptor that may lead to insulin resistance.

In my case the levels of TNFa and inflammatory cytokines was caused by sarcoidosis a autoimmune disease, this triggered the insulin resistance as i was not overweight yet.

Risk Factors
Overweight and Obesity
Main article: Central obesity

Central adiposity is a key feature of the syndrome, reflecting the fact that the syndrome's prevalence is driven by the strong relationship between waist circumference and increasing adiposity. However, despite the importance of obesity, patients that are of normal weight may also be insulin-resistant and have the syndrome.

Mine started when my sarciodosis disabled me and my weight was normal
I became Overweight because of the sarcoidosis and the co morbid disorders plus the high TNFa and inflammatory cytokines levels caused by the sarcoidosis.

Sedentary lifestyle

Physical inactivity is a predictor of CVD events and related mortality. Many components of the metabolic syndrome are associated with a sedentary lifestyle, including increased adipose tissue (predominantly central); reduced HDL cholesterol; and a trend toward increased triglycerides, blood pressure, and glucose in the genetically susceptible. Compared with individuals who watched television or videos or used their computer for more less one hour daily, those that carried out these behaviors for greater than four hours daily have a twofold increased risk of the metabolic syndrome.

You try to exercise when your feet feel like your walking on broken glass (Peripheral neuropathy caused by neurosarcoidosis) and all the major muscles in your body hurt and cramp so bad you can not sleep for days. (fibromyalgia also caused by the neurosarcoidosis )
I worked as a underground miner for 14 months this way because no doctor would give me any medication that helped. (They believed there was nothing wrong and i was drug user looking for narcotics)
then i went to doctor from UCLA through the VA because no other doctors could find anything they found the sarcoidosis that the other doctors missed, It took another 3 years to find a medication that controlled the pain.(did not eliminate it just controlled it) it did not help that the first doctors noted mistakenly that they thought i was a narcotics abuser. I am still trying to clear that up.

Even now only my Diabetes Endocrinologist believes that my weight did not cause my diabetes.
And i still have the diabetes controlled with metformin and a very low carb, high protein, high monounsaturated fat diet.

I want to add one other thing olive oil is essential for diabetics.
recipes.howstuffworks.com...

Olive oil blocks the absorption of some of the worst carbs that cause a rapid spike of blood sugar. (fast carbs)

It even blocks the effects of other bad fats if used with them.
so if you want a bowl of high fat chilli dump some olive oil in it to block the bad fat.

This is why people in the Mediterranean can eat all the high fat pork Italian sausages without high levels of heart problems and obesity
and all the high carb pasta without gaining weight.






[edit on 21-11-2009 by ANNED]

I'll star and flag for a good find and a provocative read.

However, I'll have to respectfully disagree.

Here's the problems I see:

Insulin resistance is the cause of type 2 diabetes, and it's been confirmed multiple times in clinical trials that hyperinsulinemia, through the effects high blood glucose (caused by easily digestible carbs), is causing the receptors to resist the hormone insulin.

Also, european doctors stationed in African settlements in the 50's and 60's, and the work of Weston A. Price, provided the observational evidence that supports this hypothesis. It wasn't until the introduction of easily digestible carbs (westernized diet) that these doctors began to notice the formation of diabetes, including all other diseases of civilization. Manufactured fats were not consumed.

I will ask, though, if you can provide any corroborating studies or peer reviewed work?

-Dev

Interesting. I am wondering where Hypoglycemia fits in and what, if anything, I can do about it.

You can talk to your doctor. That would be your best bet.

Edit to add: Get your fasting insulin levels checked to see if they are elevated.

[edit on 21-11-2009 by DevolutionEvolvd]

stop eating red meat, problem solved.


Mod Edit- Mod Note: One Line Post – Please Review This Link.


[edit on 22-11-2009 by elevatedone]

reply to post by ANNED

Thanks for your considered reply...

Sarcoidosis is medi-speak for an out-of-control autoimmune response caused by an imbalanced immune system...
...usually caused by dietary imbalances and/or exposure to toxic chemicals through processed foods, pharmaceuticals, personal care products and other environmental sources like pesticides or cleaning solvents.

Sounds like you are in need of a serious detox and a thorough evaluation of your diet and environmental exposure to toxic chemicals.

While western medicine have great diagnostic tools and excel at emergency proceedures...
...they are really hopeless at anything chronic.


Re Gestational Diabetes...
...there have been enormous increases recently in the frequency of gestational diabetes (400%+).

It is known that hormones produced in pregnancy block some actions of insulin, requiring increased insulin production which can trigger a diabetic crisis.

The underlying cellular changes that I have descibed in the OP affecting sugar uptake could be responsible for the increased frequency...
...because there is already an underlying predisposition...
...due to an increased stiff/sticky cell population due to poor diety fats/oils.

It has been well documented that younger people are affected by what was previously adult-onset diseases. There certainly has been an enormous increase in the consumption of short to medium chain fatty acids and corresponding decrease in consumption of the traditional fats in this age group.


The bottom line for you is…if there is evidence that Type 2 diabetes can be reversed by a change in the type of fats consumed…don’t you think it is worth exploring alternative approaches?



[edit on 22-11-2009 by troubleshooter]

Originally posted by calstorm
Interesting. I am wondering where Hypoglycemia fits in and what, if anything, I can do about it.

Hypoglycemia is often caused by Adrenal Fatigue...

This the relationship briefly...

Cortisol acts to stabilize your blood sugar...
...your adrenals produce cortisol...
...if you are deficient in some nutrients needed by the adrenals they may not produce sufficient cortisol to keep your blood sugar levels stable...
...and hypoglycemia can be the result.

Your may need nutrients that feeds your adrenals.

Originally posted by DevolutionEvolvd

Insulin resistance is the cause of type 2 diabetes, and it's been confirmed multiple times in clinical trials that hyperinsulinemia, through the effects high blood glucose (caused by easily digestible carbs), is causing the receptors to resist the hormone insulin.

Insulin resistance is caused by stiff/sticky cell membranes and compromised cell mechanisms due to the consumption of engineered food oils.


Ps I have posted the relevant section of the article mentioned in the OP below.

[edit on 22-11-2009 by troubleshooter]

The Nature of the Disease

Diabetes is classically diagnosed as a failure of the body to metabolise carbohydrates properly. Its defining symptom is a high blood-glucose level. Type I diabetes results from insufficient insulin production by the pancreas. Type II diabetes results from ineffective insulin. In both types, the blood-glucose level remains elevated. Neither insufficient insulin nor ineffective insulin can limit post-prandial (after-eating) blood sugar to the normal range. In established cases of Type II diabetes, these elevated blood sugar levels are often preceded and accompanied by chronically elevated insulin levels and by serious distortions of other endocrine hormonal markers.

The ineffective insulin is no different from effective insulin. Its ineffectiveness lies in the failure of the cell population to respond to it. It is not the result of any biochemical defect in the insulin itself. Therefore, it is appropriate to note that this is a disease that affects almost every cell in the 70 trillion or so cells of the body. All of these cells are dependent upon the food that we eat for the raw materials they need for self repair and maintenance.

The classification of diabetes as a failure to metabolise carbohydrates is a traditional classification that originated in the early 19th century when little was known about metabolic diseases or processes. Today, with our increased knowledge of these processes, it would appear quite appropriate to define Type II diabetes more
fundamentally as a failure of the body to metabolise fats and oils properly. This failure results in a loss of effectiveness of insulin and in the consequent failure to metabolise carbohydrates. Unfortunately, much medical insight into this matter, except at the research level, remains hampered by its 19th-century legacy.

Thus Type II diabetes and its early hyperinsulinaemic symptoms are whole-body symptoms of this basic cellular failure to metabolise glucose properly. Each cell of the
body, for reasons which are becoming clearer, finds itself unable to transport glucose from the bloodstream to its interior. The glucose then remains in the bloodstream, or is stored as body fat or as glycogen, or is otherwise disposed of in urine.

It appears that when insulin binds to a cell membrane receptor, it initiates a complex cascade of biochemical reactions inside the cell. This causes a class of glucose
transporters known as GLUT4 molecules to leave their parking area inside the cell and travel to the inside surface of the plasma cell membrane.

When in the membrane, they migrate to special areas of the membrane called caveolae areas. There, by another series of biochemical reactions, they identify and hook up with glucose molecules and transport them into the interior of the cell by a process called endocytosis. Within the cell's interior, this glucose is then burned as fuel by the mitochondria to produce energy to power cellular activity. Thus these GLUT4 transporters lower glucose in the bloodstream by transporting it out of the bloodstream into all the cells of the body.

Many of the molecules involved in these glucose- and insulin-mediated pathways are lipids; that is, they are fatty acids. A healthy plasma cell membrane, now known to be an active player in the glucose scenario, contains a complement of cis-type w=3 unsaturated fatty acids.17 This makes the membrane relatively fluid and slippery. When these cis- fatty acids are chronically unavailable because of our diet, trans- fatty acids and short- and medium-chain saturated fatty acids are substituted in the cell membrane. These substitutions make the cellular membrane stiffer and more sticky, and inhibit the glucose transport mechanism *

Thus, in the absence of sufficient cis omega 3 fatty acids in our diet, these fatty acid substitutions take place, the mobility of the GLUT4 transporters is diminished, the interior biochemistry of the cell is changed and glucose remains elevated in the bloodstream.

Elsewhere in the body, the pancreas secretes excess insulin, the liver manufactures fat from the excess sugar, the adipose cells store excess fat, the body goes into a high urinary mode, insufficient cellular energy is available for bodily activity and the entire endocrine system becomes distorted. Eventually, pancreatic failure occurs, body weight plummets and a diabetic crisis is precipitated.

Although there remains much work to be done to elucidate fully all of the steps in all of these pathways, this clearly marks the beginning of a biochemical explanation for the known epidemiological relationship between cheap, engineered dietary fats and oils and the onset of Type II diabetes.

* Gustavson, J., et al., "Insulin-stimulated glucose uptake involves the transition of glucose transporters to a caveolae-rich fraction within the plasma cell membrane: implications for type II diabetes", Mol. Med. 2(3):367-372 (May 1996)

Almost all diseases are now being recognised as having important abnormalities of the immune system leading to the quote "all diseases are allergic".
Obesity doesnt seem to be an exception.
www.eurekalert.org...
Perhaps obesity is just another disease related to chronic low level inflammation due to an abnormal immunogenic response to environmental antigens i.e.
1. Newly created antigens i.e. sweeteners.
2. Increased intake of old antigens i.e. added sugar and salt to processed food.
3. Increased intake of old antigens to different populations due to the international movement of people i.e. high fat/sugar/salt intake in people from the the Indian subcontinent who have moved to the West.
The reduction of the anti-inflammatory omega-3 essential fatty acids and the increase in the pro-inflammatory essential fatty omega-6 essential fatty acids in the Western diet are also contributory.

Originally posted by troubleshooter

Insulin resistance is caused by stiff/sticky cell membranes and compromised cell mechanisms due to the consumption of engineered food oils.

[edit on 22-11-2009 by troubleshooter]

How come there is not concensus or corroborating studies from other reputable researchers?

So insulin receptors aren't down regulated by the presence of excessive insulin? That seems like the more likely cause of IR.

-Dev

What happens to the hormone made from visceral fat?? It must effect the cell or liver in some way??I always thought a high carb / fructose diet causes insulin resistance?

If you have some genetic trait that causes the liver to store fat instead of burn you now have more visceral fat.Could this effect cells and then lead to insulin resistance??A high carb omega 6 fructose diet would compound insulin resistance & fatty liver.

reply to post by fishing


Leptin is a hormone produced by fat cells that affects the hypothalamus, the thyroid and, ultimately, metabolism.

Some researchers have found that leptin resistance can form, much like insulin resistance, when excessive amounts are present in the blood. These high levels are almost always caused by, or at least associated with too much body fat.

The thought is that it's possible leptin resistance is inhibiting the signal that makes one stop eating, which leads to overeating...and obesity...and more leptin.

Leptin resistance is thought to be directly attributed to carbohydrate consumption, just as insulin resistance is.

The problem with this is; A) Research is limited and relatively new, B) It completely ignores the fact that overeating doesn't cause fat deposition but that Insulin regulates fat metabolism, C) Most of the data are based on mutant mice research.

-Dev

Originally posted by DevolutionEvolvd

Originally posted by troubleshooter

Insulin resistance is caused by stiff/sticky cell membranes and compromised cell mechanisms due to the consumption of engineered food oils.

[edit on 22-11-2009 by troubleshooter]

How come there is not concensus or corroborating studies from other reputable researchers?

I spent some time reviewing the medical literature...
...there is plenty of research regarding the role of GLUT4 transporters and the role cis-type w=3 unsaturated fatty acids...
...but all of those I reviewed were concerned with finding unique therapuetic mechanisms to exploit various pathways for the production of active substances as pharmacuetical agents.

This should be expected as most research is funded by companies searching for unique agents for this purpose.

The bottom line for me is this...
...Thomas Smith came to the conclusion in the OP while searching for the cure of a personal condition...
...his approach from 'first principles' successfully cured his condition...
...I have observed similar results when his method was applied to other people.

Eliminating engineered fats/oils (those extracted using heat and chemical methods)...
...and substituting with a small amount of cold pressed Flax or other unrefined edible oil works.

If you have been diagnosed with Type 2 diabetes isn't it worth trying?



[edit on 28/11/09 by troubleshooter]

Here is the link to the paper referenced in the OP...

Joslyn, E.P., Dublin, L.I., Marks, H.H., "Studies on Diabetes Mellitus", American Journal of Medical Sciences 186:753-773 (1933)

www.ncbi.nlm.nih.gov...

Changes in the cell due to the consumption of engineered 'edible' oils is the cause of Type 2 Diabetes.

reply to post by troubleshooter


The majority of type 1 diabetes comes from vaccines, which suits the Pharms just fine, as type 1 makes them big money