It looks like that there is a specific characteristic to the viral attack: The more hyperactive the immune system is, the more dramatic is the outcome
- more fatalities. Kids have a very strong immune response…
Although it is always mentioned, that there were “underlying health conditions” in the infected kids in question, this looks like a smoke-screen
to me. It is maybe supposed to hide that the more healthy you are, the more likely you are going to die.
.. which would be very bad news, because it would mean that the common avenue to boost the immune system will only result into a higher probability to
So, what to do? Vaccination, some will say. But it looks like as if vaccination does not affect the new strain, the vaccine fails (see link above).
Vaccination would make things even worse, because the vaccine “boosts” also the immune system, through the toxic squalene. Then the hyperactive,
“boosted” immune system runs into the virus, while being affected by the squalene poisons – a fatal double whammy. Maybe that’s why they are
so interested to get us all vaccinated, even the (humane?) CFR people…
I hope that I am wrong, and if so, then please post and correct me.
But if not, what to do?
It looks like a typical CATCH 22 situation. (I really like the film)
It is very difficult to find data on that in the internet. It looks like there is a general consensus throughout the international medical community
NOT to disclose medical details. This looks not like an organized gag-order, but has obviously more to do with a (only human) response to the
FRIGHTENING NATURE OF THE DISEASE. But the info that seeps through is just terrifying:
The mutant virus goes deep into the lung and starts there its destructive work. Doctors will often do only a nasal swab for testing. While the
patient will test negative for the virus, it is busy down in the lung.
So what is happening in detail: The mutant virus can survive higher temperatures and is therefore going deep into the lungs into the area of the fine
Alveoli, where the oxygen-to-blood-transfer is done via a very thin membrane:
Lets look at the hypothetical case of Mr. John Doe:
The “prepared” Mr. Doe is walking the street, thoroughly protected by tight clothing, gloves and a really good filter-mask. A microscopic drop,
containing the virus, attaches itself to any fine tissue at the eyes. The virus enters the body of Mr. Doe through this gate.
The mutant virus travels deep into the lung and starts there its destructive work. The infection of Mr. John Doe starts in the beginning like an
ordinary flu-infection: Fever, maybe nausea etc..
Mr. John Doe will enter a
Systemic inflammatory response syndrome (SIRS).
(SIRS, I don’t like you !)
Mr. Doe feels that something is not right, and as a precaution gets himself tested for swine flue. The doctor will do only a nasal swab for testing.
And while Mr. Doe will test negative for the virus, it is busy down in the lung. The mutant virus can survive higher temperatures and is therefore
going deep into the lungs into the area of the fine Alveoli, where the oxygen-to-blood-transfer is done via a very thin membrane.
The immune systems white blood cells will react to this infection by releasing Cytokines (a messenger substance), which will attract more white
blood-cells to fight the virus. These cytokines-releases are normally well regulated, just enough to call for the help required.
The patient Doe enters the stage of SEPSIS.
Up to now everything is normal, but then….
In this case there is a massive pathogenic attack on the way. So something somehow goes wrong:
- Either the white cells fail to initiate a pathogenic recognition of the virus, which would start the fight against it. Instead the cells will call
for more help by releasing more cytokines.
- Or the onslaught is so massive that it somehow triggers an unproportional (extreme and massive) cytokine response from the white cells. For that the
virus has to have an extremely high rate of reproduction, which might be the case here.
Anyway, the result is: An ongoing massive overproduction of cytokines, with no check and balance in place. The mechanism that triggers this response
is not clear. It is called a CYTOKINE STORM (CS).
What is a cytokine storm and how severe are the symptoms?
In 2006 a new substance called TGN1412 was tested, which caused a CS in the test persons. Here is what happened:
By now the patient Doe will have entered the next phase: Septic Shock
As the cytokine-production goes over the roof, even a layman can now recognize that something goes very wrong. The cytokines opens the blood vessels,
so that plasma and blood can enter into the body-texture and –organs. The thin membrane of the Alveoli becomes porous along with other membranes.
The blood pressure will sink rapidly, because of the capillary leakages.
Sometimes an “acute alteration in mental status” of the patient is observed. In other words, the patient faints and/or gurgles nonsense.
Now the able doctors will recognize that the patient Doe has entered the phase of a
CYTOKINE STORM (CS)
Systemic Capillary Leak Syndrome (SCLS)
Patient Doe will be admitted to ICU.
But now the lungs have started filling up with fluids:
Please notice the dark areas at the periphery. Thats were the Alveoli are.
And within only 6 hours it will look pretty bad:
The dark areas are liquids in the lungs.
The blood pressure collapses because of the drain of fluids.
To stabilize the blood pressure, the patient Doe will receive (a lot of) fluid by intravenous means. But that fluid will immediately leak out into the
body-organs and -tissue. As the patient will receive up to 40 liters of fluid (or more), the body will swell and become unrecognizably bloated. At
that stage some Does will go into cardiatic arrest, but not our Doe. He is already unconscious, but his body still fights.
Depending on the scope of the capillary leakage the lungs will be filled with either blood-plasma or blood-cells plus plasma.
This pumping-up of the whole body inhibits the flow of oxygen and nutrition to the vital organs (Ischemia). Patient Doe will of course get help
through a respirator. And while others will die of intrapulmonary haemorrhage necrotizing pneumonia, our Doe is still on it. As the lungs are no more
functional and the CS is still running, the patient Doe will now enter into the stage of
Multiple Organ Dysfunction Syndrome (MODS)
The kidneys, lungs, heart, liver etc. are starting to fail. That’s where the story of our patient Doe ends fatally.
But wait, I forgot: As the CS is an immunological reaction to an infection which can not be overcome by conventional means, the doctors will give the
patient immunosuppressors in order to overcome at least the unregulated cytokine production. The result is a very weak, bloated patient with a
suppressed immune system and a multiple punctured body. It’s where secondary infections like Staphylococcus Aureus et al are entering the playground
to finish up the undone.
It’s a horrible death.
So this is a CYTOKINE STORM. So, to be clear: Whatever is done, either the CS hits Doe or the secondary infections. A real CATCH.
Oh sorry, I forgot to nail the coffin. As posted earlier:
Quote: “Because cytokine inhibition does not protect against death, therapies that target the virus rather than the cytokines may be preferable.”
It means that even if patient Doe survives the CS, he still has to get rid of the fast multiplying virus.
It’s a real CATCH 22.
It’s a horrible death.
SO LETS FACE THE TIGER :
Here a pic of a fatal case of SCLS/MODS. WARNING: GRAPHIC IMAGE!!
The light bluish skin is a result of anaemic blood, blood without oxygen. It’s the Cyanosis Blue of 1918.
You can see the blood in the lungs protruding from the mouth.
That’s the answer to why we have a mum throughout the medical community. The truth is so devastating, that nobody wants to admit it in public.
Yes folks, as cruel as it seems: That’s what some of us are in for…
Still thinking about sauerkraut and a good digestive rest?