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According to a 1995 report in the journal Nature Genetics, a mother brought her infant boy to the doctor after noticing that his head was developing abnormally. When doctors analyzed his blood, they found something truly bizarre: Despite his anatomically male features, the boy's blood cells were entirely female, consisting only of genetic material from his mother. Some of his other cells—such as those found in his urine—were normal, consisting of a combination of both maternal and paternal DNA. No one knows exactly how this occurred, but the best guess is that immediately after being fertilized, one of his mother's eggs fused with a neighboring unfertilized egg that was dividing parthogenetically. This gave rise to a boy who was considered half-parthenogenetic, since approximately half of his cells were derived from a "faux" conception, containing no remnants of his father's DNA.
n mice, parthenogenetic embryos die at the early postimplantation stage as a result of developmental requirements for paternally imprinted genes, particularly for formation of extraembryonic tissues. Chimaeric parthenogeneticharrnormal mice are viable, however, due to non−random differences in distribution of their two cell types. Species differences in imprinting patterns in embryo and extra−embryonic tissues mean that there are uncertainties in extrapolating these experimental studies to humans. Here, however, we demonstrate that parthenogenetic chimaerism can indeed result in viable human offspring, and suggest possible mechanisms of origin for this presumably rare event.
Originally posted by Happyfeet
I dont understand, the egg was still fertilized correct?