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Two key memorandums from WHO, discovered by Patrick Jordan, prove WHO has intentionally created the three-shot killer vaccine that people in the USA and other countries could soon be forced to take.
1972 WHO Bulletin 47, No 2 Memordanda #1 and #2 Virus-associated immunopathology:
Animal models and implications for human disease * technically outline the ability to create biological weapons in the form of vaccines that:
1) First totally disable the Immune System.
2) Load every cell of the Victim’s body up with Infection.
3) Switch the Immune System on causing the host to kill themselves in a Cytokine Storm.
One, Two, Three, Dead.
These WHO Memorandas describe the three-stage impact of the three "shots" many people will be forced to take this fall to allegedly treat a virus that WHO also helped create and release.
This is a crucial piece of evidence of WHO's long-term genocidal intentions that could stand in any court of law because these memorandums give the best and fullest explanation WHO's and affiliated labs (such as the CDC) current activities, such as their patenting of the most lethal bird flu viruses, their sending that virus to Baxter's subsidiary in Austria, which weaponised it and sent out 72 kilos to 16 labs in four countries almost triggering a global pandemic.
For every crime, there needs to be motive, an indication that it was deliberate, planned. The WHO memorandums provide the evidence of just that deliberate, long-term planning to kill people by weakening their immune system by use of the first vaccine, injecting a live virus into their body by a second, and creating a cytokine storm using squalene in a third.
Virus-associated immunopathology: animal models and implications for human disease: 1. Effects of viruses on the immune system, immune-complex diseases, and antibody-mediated immunologic injuryBull World Health Organ. 1972; 47(2): 257–264. PMCID: PMC2480894| Summary | Page Browse | PDF–1.2M | Virus-associated immunopathology: animal models and implications for human disease: 2. Cell-mediated immunity, autoimmune diseases, genetics, and implications for clinical researchBull World Health Organ. 1972; 47(2): 265–274. PMCID: PMC2480896| Summary | Page Browse | PDF–1.5M |
The tissue damage caused by virus infection has been traditionally explained by the ability of viruses to multiply in cells and thereby injure or destroy them. Recent evidence suggests, however, that lesions may also be caused by the host's immune response to viral antigens and that the immune system itself may be perturbed by some viruses. This memorandum reviews recent developments in viral immunopathology, with special reference to animal model systems, and indicates the possible relevance of the new concepts and techniques for certain diseases of man. Certain viruses, notably the leukaemia viruses and some of those causing persistent infections, depress the host's ability to mount an antibody response to antigens, while other viruses may enhance the antibody response. Cell-mediated immunity may also be depressed. Another immunopathological manifestation of virus infection is immune-complex disease. When viruses or their antigens persist in the circulation they combine with specific antibody, and the resulting complexes lodge in various sites, especially the kidney. Further combination with complement leads to the release of tissue-damaging substances. A third condition associated with virus infection is antibody-mediated immunologic injury. Both oncogenic and non-oncogenic viruses frequently induce new antigens on the surface of the cells they invade. When antibody attaches to these antigens in the presence of complement, the cells are destroyed.
Part 2 of this memorandum describes further mechanisms whereby the interaction of a virus with the host's immune system may lead to tissue damage. Cell-mediated immunity plays a vital role in promoting recovery from virus infections, but under some circumstances tissue damage may be caused by the reaction of immune cells with viral antigens. When mice are infected with lymphocytic choriomeningitis virus neonatally or as adults while receiving immunosuppressive drugs, widespread invasion of cells is seen but there is little overt disease. If, however, normal adults are infected or if immune cells are transfused into tolerant mice, cell injury and death follow. Viruses have long been suspected of contributing to the pathogenesis of autoimmune diseases. Antibodies directed against normal cell constituents have been reported in several virus infections. Viruses may conceivably unmask or release host antigens, alter host antigens and act as “helper determinants”, or perhaps in other ways provoke immune responses against normal body constituents. The immunopathological manifestations caused by viruses may also be influenced by the host's genetic makeup. Certain observations indicate that, in addition to controlling susceptibility to virus infection, genetic factors partly determine the effectiveness of the immune response. The memorandum calls attention to the possible implications of these concepts and findings for clinical research. Some of the diseases of animals and man that serve as models for studies of virus-associated immunopathology are briefly described.
Originally posted by unicorn1
Where does it say about using VACCINES to do this?
Or is the title of the thread supposition, not fact?
And no I'm not an 'disinfo agent' so don't give me any of that old crock. I just check on sources.
He acknowledged there may be an "emotional response" from critics who argue that terror suspects should not be allocated swine-flu medications while members of the U.S. public are still waiting due to a vaccine shortage.
But he said U.S. military officials are "responsible for the health and care of the detainee population."