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Originally posted by L.HAMILTON
Used to be if you wanted to eat healthy, it was one of the cheapest grocery bill buying nothing but vegetables and food that is good for you. Now the tables are reversed, if you want to go on a diet it will cost twice as much as it used to. In today's economy people have no choice but to feed their families the cheaper more fattening foods. No, if we are looking at who to blame, I would say the FDA, allowing food Co. to add more fat and non healthy ingredients to their food products.
Originally posted by NoJoker13
reply to post by DevolutionEvolvd
Then why is America more over weight than every other country?
Are you meaning to tell me that we're the only place where this predisposition is found?
If so elaborate on your thesis of how Americans can be so over weight (at about the rate of 61% of adults), when the rest of the worlds highest obesity rate only hites 50% of adults(this is only in Russia)? So clearly there is a trend in America that seems to be "spreading" to other countries. Sorry it's no disease, but be my guest thats the easiest way out.
Originally posted by DevolutionEvolvd
reply to post by KrazyJethro
I understand what you're saying. However, you did find your way to this website in a search for knowledge so it's only predictable that you would research all you can to further improve your health. Not everyone has that drive. You're in the minority.
Considering the amount of nutritional/health information in bookstores and on the net, how does one make a final choice as to what's the healthiest advice?
Even nutritionists and dietitians have a huge misunderstanding of biochemistry and nutrient biology. Who do you believe if you're venturing into the confusing world of nutrition science for the first time?
Originally posted by xenchan`
well i read alot of post and i wish i could get that cheap with food a month its cheaper ware i live to buy the prepackaged stuff then it is stuff to make something bu scratch
The primary markers of the metabolic syndrome are central obesity, insulin resistance and hypertension. In this review, we consider the effect of changes in maternal nutrition during critical windows in fetal development on an individual's subsequent predisposition to the metabolic syndrome. The fetal origins of obesity, cardiovascular disease and insulin resistance have been investigated in a wide range of epidemiological and animal studies; these investigations highlight adaptations made by the nutritionally manipulated fetus that aim to maintain energy homeostasis to ensure survival. One consequence of such developmental plasticity may be a long term re-setting of cellular energy homeostasis, most probably via epigenetic modification of genes involved in a number of key regulatory pathways. For example, reduced maternal–fetal nutrition during early gestation to midgestation affects adipose tissue development and adiposity of the fetus by setting an increased number of adipocyte precursor cells. Importantly, clinically relevant adaptations to nutritional challenges in utero may only manifest as primary components of the metabolic syndrome if followed by a period of accelerated growth early in the postnatal period and/or if offspring become obese.
Two children considered to be at risk of abuse because they are severely obese have been removed from the protection register after scientists discovered that they carry a newly identified genetic abnormality that explains their weight.
Evidence from a ground-breaking study has convinced social workers that the children’s obesity was not caused by parental neglect or deliberate overfeeding but by a missing segment of DNA. The cases of another two children on the at-risk register have also been placed under review, after research showed them to have the same genetic deletion.