Sat Sep 19, 11:14 pm ET
CHICAGO – The University of Chicago Medical Center says the infection that killed a scientist may be connected to bacteria he researched that causes
The university said Saturday that its researcher studied the genetics of harmful bacteria including Yersinia pestis, which causes the illness. He died
Sept. 13. His name and age haven't been released.
The medical center says the bacteria he worked with was a weakened strain that isn't known to cause illness in healthy adults. The strain was
approved by the Centers for Disease Control and Prevention for laboratory studies.
An autopsy found no obvious cause of death but did find the presence of the bacteria. More tests are planned. No other illnesses have been
Now here is a link to what wiki says aabout Yersinia pestis
I've seen other stories on the web that say he died from the plague bacteria.
Apparently he was a healthy adult or he would of been a fool to even be around this bacteria. Yet the cdc said is wasn't known to cause illness in
Pathogenics and immunity
In the urban and sylvatic cycles of Y. pestis most of the spreading occurs between rodents and fleas. In the sylvatic cycle the rodent is wild, unlike
in the urban cycle, where the rodent is domestic. Additionally Y. pestis can spread from the urban environment and back again. Every infected animal
can transmit the infection to humans through contact with skin tissue. Humans can also spread the bacteria to other humans through sneezing, coughing
or direct contact with infected tissue.
If this man did die from the plague bacteria how long was he walking around in public contaminating other people?
If you read the wiki article some of this type bacteria gives symptoms much like the swine flu does. Fever, cough sneezes. So you go to the doctor
thinking you have the swine flu they treat you for the swine flu. The swine flu treatment will do no good on this bacteria.
I've been told that anyone coming to the doctors with flu symptoms are being treated for the swine flu without testing.
Without that testing how would they know if you had the swine flu or you might of been exposed to the bacteria?
A formalin-inactivated vaccine once was available for adults at high risk of contracting the plague until removal from the market by the U.S. Food and
Drug Administration. It was of limited effectiveness and may cause severe inflammation. Experiments with genetic engineering of a vaccine based on F1
and V antigens are underway and show promise; however, bacteria lacking antigen F1 are still virulent, and the V antigens are sufficiently variable,
that vaccines composed of these antigens may not be fully protective. United States Army Medical Research Institute of Infectious Diseases
(USAMRIID) have found that an experimental F1/V antigen based vaccine protect cynomolgus macaques, but fails to protect African green monkeys. A
report found that Europeans were less likely to catch the plague, because they are the descendants of the survivors of the plagues that affected
Europe in the medieval times
The traditional first line treatment for Y. pestis has been streptomycin, chloramphenicol, tetracycline, and fluoroquinolones. There is
also good evidence to support the use of doxycycline or gentamicin. Resistant strains have been isolated; treatment should be guided by antibiotic
sensitivities where available. Antibiotic treatment alone is insufficient for some patients, who may also require circulatory, ventilator, or renal
In an emergency department setting, Harrison's Principles of Internal Medicine outlines the following treatment course. Antibiotics within the
first 24 hours is very beneficial, with intravenous being preferred in pulmonary or advanced cases. Streptomycin or gentamicin are the first-line
drugs, with chloramphenicol for critically ill patients, or rarely for suspected neuro-involveme
I have to wonder how a harmless bacteria could kill a healthy person. Unless that persxon didn't have the harmless strain of the bacteria.
This comes from wiki also. I'll assume it's speaking of the same man as the article from the ap.
On September 13, 2009, Malcolm J. Casadaban, a University of Chicago molecular genetics professor, who was doing research involving Yersinia pestis
died and his death later was determined related to this bacteria, possibly contracted in his own lab..
Recently Y. pestis has gained attention as a possible biological warfare agent and the CDC has classified it as category A pathogen requiring
preparation for a possible terrorist attack.
What if the swine flu is not the real killer but a smoke screen? The government could defend their actions by saying we were fighting a pandemic of
flu we had no way of knowing this would come along also.
If I have this in the wrong catagory sorry and thank you for putting it where it belongs.