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EXPOSURE TO AERIAL EMISSIONS OF NANO COMPOSITE MATERIALS RESULTED IN CHOLINESTERASE INHIBITION
by Hildegarde Staninger, Ph.D., RIET-1, Industrial Toxicologist/IH & Doctor of Integrative Medicine. Integrative Health Systems, LLC, 415 3/4th N. Larchmont Blvd., Los Angeles, CA 90004 Tel: 323-466-2599 Fax: 323-466-2774 e-Mail: ihs-drhildy@sbcglobal.net © September 7, 2009
Presented at the 2009 National Registry of Environmental Professionals Conference Oct 5-6th, 2009, Des Plaines, IL. Published NREP Journal of Environment and Sustainability.
ABSTRACT: Cholinesterase inhibition has been associated with exposure to oranophosphate and carbamate pesticides, since their creation during World War I and II. Traditionally, they cause severe neurological disorders that can paralyze not only insects but humans. Individuals are usually exposed by misuse of pesticide applications or through aerial spraying.
The cholinesterase inhibition effects for chronic exposure or multiple acute exposures are expressed in acetyl cholinesterase, enzyme (AChE) activities inhibition. The integration of nano particulates in various nano composite materials, such as aerial hydrogels and other similar materials used in aerial vector sprays, weather modification and sensor grids, resulted in the individual having as high as 96.2 % cholinesterase inhibition and detectable readings of nano composite materials present. Confirming Ocean University, China’s findings in the June 2009 issue of Chemical Sensitivities, that nano particles are 100 times more toxic than a single molecule of a pesticide, such as Malathion, propoxopur or benomyl.
Background: During the summer of 2007 in Phoenix, Arizona, a female age 51 was exposed to aerial spraying for weather modification and vector control (mosquito) as performed through G-1 requirements and Project Earth Scope1. She was at home and had her windows open and went outside of her home to see what was happening. She immediately felt a burning and tingling sensation and became ill.
The duration of symptoms depends partly on the rate of AChE reactivation. Spontaneous reactivation depends on the chemical structure attached to the enzyme. The reactivation of the inhibited enzyme can be facilitated considerably by special compounds (oxymes).
Several of these compounds have become important antidotes in the treatment of pesticide poisonings. The inhibition enzyme may also be transformed into a state where no spontaneous reactivation occurs and where oximes are no longer capable of reactivating it.
The phenomenon is called “aging” and is characterized by removal of one of the alkyl groups form the phosphoryl groups attached to the enzymes. The rapidity of inhibited AChE aging depends on the chemical nature of the phosphorylating materials. It must be noted that in the materials previously found by other patients exposed to advance nanomicrobic materials/nano particles phosphorus was present in other exposed individuals.
Over the last year the individual has experienced the following classical AChE symptoms with the appropriate site of action (target organ system): Site of Action Signs and Symptoms Eyes Increased lacrimation, slight myosis (occasional unequal, later marked, blurred vision, eye pain when focusing, frontal headaches, conjunctive hyperemia.
Respiratory System Rhinorrhea, hyperemia (local exposure), tightness in chest, Prolonged wheezing, bronchoconstriction, increased secretion, dispnea (not enough air), slight chest pain, cough, edema of the lung.
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[edit on 19-12-2009 by spinkyboo]
[edit on 19-12-2009 by spinkyboo]
