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They proved their hypothesis by showing that regardless of the taste the rats were trained to avoid, they forget their learned aversion after a single application of the drug.
The technique worked as successfully a month after the memories were formed, which is analogous to years in humans, and all signs so far indicate that the affected unpleasant memories of the taste had indeed disappeared.
They hypothesized, that this protein, an enzyme located in the synapses called PKMzeta, acts as a miniature memory “machine” that keeps memory up and running by changing some facets of the structure of synaptic contacts. But it must be persistently active to maintain this change, which is brought about by learning.
Originally posted by djohnsto77
Well we already know drugs and alcohol can prevent long-term memories from forming. But this seems to be something different, a drug administered after the memory was formed that deleted it. My main question is the specificity of this. Does it remove all memories, or can it be targeted to specific ones?