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originally posted by: network dude
a reply to: bastion
do you inject it, or just rinse your mouth with it?
originally posted by: network dude
do you inject it, or just rinse your mouth with it?
originally posted by: Cymru
a reply to: bastion
I just read this on the local rag.
We've been told you can contract the virus through eyes, nose and mouth.
Are we expected to snort and eyerdop the suff as well? (/sarcasm)
www.walesonline.co.uk...
originally posted by: network dude
a reply to: bastion
do you inject it, or just rinse your mouth with it?
originally posted by: OtherSideOfTheCoin
Trump “I hear injecting mouthwash can cure the virus”
originally posted by: Jubei42
I don't see the value of this discovery, it's a surface desinfectant. There are many of them. How does that help with an airborne virus?
originally posted by: Jubei42
I don't see the value of this discovery, it's a surface desinfectant. There are many of them. How does that help with an airborne virus?
Emerging studies increasingly demonstrate the importance of the throat and salivary glands as sites
of virus replication and transmission in early COVID-19 disease. SARS-CoV-2 is an enveloped virus,
characterised by an outer lipid membrane derived from the host cell from which it buds.
While, it is highly sensitive to agents that disrupt lipid bio-membranes, there has been no discussion about
the potential role of oral rinsing in preventing transmission. Here, we review known mechanisms of
viral lipid membrane disruption by widely available dental mouthwash components that include
ethanol, chlorhexidine, cetylpyridinium chloride, hydrogen peroxide and povidone-iodine. We also
assess existing formulations for their potential ability to disrupt the SARS-CoV-2 lipid envelope,
based on their concentrations of these agents, and conclude that several deserve clinical evaluation.
....
It is becoming increasingly recognised that the throat is a major site of replication and shedding of
virus in COVID-19 illness, and that viral load is important(25). Throat and sputum are abundant in
particles, which peak 5-6 days after symptom onset, and decline thereafter(26, 27).
Viral load
correlates with older age(27), and a study of 76 patients in Nanchang, China, showed that those
with severe SARS-CoV-2 tend to have higher viral load and longer virus-shedding period than those
with mild disease(28). Similarly, viral load was linked with lung disease severity in a study of 12
patients with pneumonia(29). Many asymptomatic individuals have modest levels of detectable
viral RNA in their oropharynx for at least 5 days, which is similar to individuals with clinical
symptoms(30). Data from GTEX gene expression data indicates that ACE2 (a key receptor for COVID19) expression is higher in salivary glands than lungs, suggesting that the these could be a major
source of new viral particles(31).