It looks like you're using an Ad Blocker.
Please white-list or disable AboveTopSecret.com in your ad-blocking tool.
Thank you.
Some features of ATS will be disabled while you continue to use an ad-blocker.
originally posted by: Phantom423
As per usual, Coop has gone into hiding. When you can't face the evidence, just ignore it. Works every time - for him anyway.
originally posted by: Phantom423
As per usual, Coop has gone into hiding. When you can't face the evidence, just ignore it. Works every time - for him anyway.
Is there an obsessive psychosis? Aetiological and prognostic factors of an atypical form of obsessive-compulsive neurosis. Solyom L, DiNicola VF, Phil M, Sookman D, Luchins D.
Abstract The study explores whether an atypical form of obsessional illness can be delineated and separated from the conventional form of obsessive-compulsive neurosis (OCN). From a group of 45 obsessive patients, 8 were selected on the basis of 3 criteria: presence of a severely debilitating main obsessive symptom; bordering on the delusional; no schizophrenic symptoms. Assessment and outcome measures included the Psychiatric Questionnaire, the Leyton Obsessional Inventory, Fear Survey Schedule, and IPAT Self-Analysis Form. Self-assessment forms allowed patients to make social adjustment and neurotic symptom ratings. In a multimodal approach, patients were assigned to behavioural and pharmacological treatments on the basis of severity. Reassessment took place after 50 sessions of therapy. Results of analysis of variance statistics indicated that the atypical group had a more malignant form of illness, with more varied and severe obsessions. A poorer prognosis for the atypical group was indicated by: greater social maladjustment, poor employment records, illness of longer duration showing no remissions despite more courses of treatment, and poor response to treatment throughout. The atypical group manifested fewer characteristic features of OCN (example: fewer precipitating events). On the other hand, schizophrenia was not imputed, although delusion-like experiences in the atypical group suggest a psychotic form of illness. The term "obsessive psychosis" suggested by Strauss and recently investigated by Weiss et al and Robinson et al is proposed for our atypical group. Results are compared with those of other investigators. It is concluded that the delineation of a subgroup of obsessional illness is desirable for research and therapy since a form of atypical obsessional illness or obsessive psychosis can be differentiated on aetiological, phenomenological and prognostic factors.
I honestly don't think you guys even know what you believe. PhotonEffect is an exception, because she/he can actually articulate their beliefs and offer information in their own words.
originally posted by: Phantom423
a reply to: peter vlar
Coop wouldn't dare do that. He'd be flattened as if an 18 wheeler ran over him. It's tactical stupidity - by saying nothing is on the table when the table is full makes him feel superior - he knows something the rest of us don't know. He exhibits the psychotic behavior outlined in the article below - obsessive, delusional, separated from reality.
originally posted by: Noinden
a reply to: cooperton
If it were epigenetics, and only epigenetics, one would not be able to explain it away as an SNP mutation (which it is) and that there are variations of it.
Stop making things up.
originally posted by: Noinden
a reply to: cooperton
Again you are not reading what I aasked. You said it was the primary mode. Prove it.
BURDEN
OF
PROOF
etc
originally posted by: Noinden
a reply to: cooperton
Again you are not proving anything, you are making statements, based on your own personal bias. Science requires more. It is science we are talking here, not your beliefs.
originally posted by: cooperton
originally posted by: Noinden
a reply to: cooperton
Again you are not proving anything, you are making statements, based on your own personal bias. Science requires more. It is science we are talking here, not your beliefs.
1) Epigenetic mechanisms are the primary modulator of all genes.
That one you got right, bravo!
2) Lactase is a gene
3) Therefore, epigenetic mechanisms are the primary modulator of the lactase gene.
There are already studies demonstrating this:
"However, an epigenome-wide approach using the Illumina Infinium HM450 bead chip identified a differentially methylated position in the LCT promoter where methylation levels are associated with the genotype at −13910C > T, the persistence/non-persistence phenotype and lactase enzymatic activity"
Study 2018
"epigenetically controlled regulatory elements... accounted for inter-individual differences of lactase mRNA level in a Lithuanian cohort of individuals"
Study 2017
" Epigenetically-controlled regulatory elements were found to account for the differences in lactase mRNA levels between individuals, intestinal cell types and species."
study 2016
Genetic factors contribute to epigenetic changes occurring with age at the regulatory elements, as lactase persistence- and non-persistence-DNA haplotypes demonstrated markedly different epigenetic aging. Thus, genetic factors facilitate a gradual accumulation of epigenetic changes with age to affect phenotypic outcome.