OP/ED: The Final Solution, page 3

Forget all this population junk. I say us regular people need to combine our resources, team up with a few scientists, and create our own nation out in International waters. How do I propose we do this? Im glad you asked. Although I may be young and naive the concept of my idea is definetly possible.

My theory is we could build aqua cities that would resemble that of oil rigs. Since we can already build a structure as grand as an oil rig it should not be too hard to build one made as a colony. If we build them against certain ocean currents we could use the dam concept for hydro electric power. I believe with the proper research this is possible. As well we could probably have wind turbines dotting the area because the open ocean has plenty of uninterrupted wind! Can you imagine harnessing the wind power from a tropical storm or even a hurricane!! (assuming our wind turbines are sturdy enough to withstand the force) We could have built in desalination to treat the water and build platforms to grow genetic crops quickly and in vast quantities. Of course it sounds like fantasy but if this kind of research were given the proper budget and research I believe a city like this can exist within the next 20-30 years.

Only a dream I suppose...

But hey if were looking to control the population I say forget waiting 10 years for further testing on those male contraseptive pills just start handing them out in India, China, and Africa and let it be tested like that

A disease called fibromuscular dysplasia (FMD) appears to be the original infectious prion strain, introduced into the human population in the early 1900’s. It’s emergence is linked to the world’s first mass-vaccinations, of American soldiers for WWI. FMD finally was acknowledged officially in 1938, in the USA, when it was found to have been transmitted congenitally. The disease involves actin proteins mis-folding into “a-smooth muscle actin” (a-SMA). The presence of mis-folded a-SMA protein indicates that FMD is caused by an infectious prion.

The medical literature is rife with myth, spin and contradictory dis-information about FMD. A superficial search reveals that FMD is a rare disease, yet at the same time, a ‘not uncommon’ pathological finding, usually benign; it is also described as a natural consequence of aging and a genetic disorder – that primarily affects women.

The truth about FMD is buried just below the surface, distributed throughout the US National Institutes of Health PubMed database. PubMed references millions of medical and research articles on FMD, dating back to the early 1950’s.

For example: FMD was found in domestic turkeys in 1980 and then in steelhead trout in 1981, both in the USA. By 1996, FMD was infecting quail in Japan. So according to official spin, animals and human females get FMD, but men are immune. Like, the disease is gender-specific in humans and transmitted genetically from humans to fish and birds. Sure. Wanna buy a bridge?

* “The occurrence of fibromuscular dysplasia in the arteries of domestic turkeys.” Julian LM. Am J Pathol. 1980 Nov;101(2):415-24. PMID: 7435545
* “The ultrastructure of spontaneous coronary arterial lesions in steelhead trout (Salmo gairdneri).” House EW, Benditt EP. Am J Pathol. 1981 Sep;104(3):250-7. PMID: 7294154
* “Fibromuscular dysplasia in intramuscular arteries of Japanese quail (Coturnix coturnix japonica).” Braga IS 3rd, Tanaka S, Itakura C, Mizutani M. J Comp Pathol. 1996 Feb;114(2):123-30. PMID: 8920213

FMD usually takes decades to progress unless something triggers rapid progression, such as exposure to high-level environmental contamination. Symptoms are not verified clinically and FMD generally is not diagnosed until victims are older, and the disease has progressed. By the time men hit their 40’s, their FMD usually has morphed into atherosclerosis. It only looks like men don’t get FMD.

* “All forms of fibromuscular dysplasia are progressive and have variable rates of progression.”
Radiology, Vol 139, 45-51, Copyright © 1981 by Radiological Society of North America. Progression of renal artery fibromuscular dysplasia in 42 patients as seen on angiography. V Goncharenko, AJ Gerlock Jr, MI Shaff and JW Hollifield.
radiology.rsnajnls.org...

* “Fibromuscular dysplasia is a chronic progressive disease and some cases progress rapidly in a few months.”
Angiology. 1999 Feb;50(2):153-6. Fibromuscular dysplasia involving coronary arteries--a case report. Ogawa T, Nomura A, Komatsu H, Fujii S, Urasawa K, Okamoto H, Kitabatake A. Department of Cardiovascular Medicine, Hokkaido University School of Medicine, Sapporo, Japan. PMID: 10063947
* “Age and fibroplasia as preconditions for atheronecrosis in human coronary arteries.” Arch Pathol Lab Med 1987 Oct;111 (10):957-63 Tracy RE, Kissling GE. PMID: 3632271
* “Evolution and progression of atherosclerotic lesions in coronary arteries of children and young adults.” Arteriosclerosis. 1989 Jan-Feb;9(1 Suppl):I19-32 Stary HC. Louisiana State University, School of Medicine, New Orleans 70112. PMID: 2912430

For decades, various scientists, researchers and health activists have been trying to tell authorities and the world about FMD.

For example:

“Fibromuscular dysplasia in a child: a generalized arterial disease.” Acta Paediatr Scand 1980 Jul;69(4):563-6. Pesonen E, Koskimies O, Rapola J, Jaakelainen J. PMID: 7446106

“Fibromuscular dysplasia may be a systemic disease…, and may affect the central nervous system.”
Renovascular hypertension and demyelimating disease in a young woman. Alaska Med 1989 Jul-Dec;31(4):137-43. Neubauer RL. PMID: 2596648

“This report suggests that FMD should be considered to be a systemic angiopathy…”
“Clinical significance of cerebral aneurysm in renovascular hypertension due to fibromuscular dysplasia: two cases in siblings.” Angiology 1989 Jun;40 (6):581-8. Ouchi Y, Tagawa H, Yamakado M, Takanashi R, Tanaka S. Department of Internal Medicine, Mitsui Memorial Hospital, Tokyo, Japan. PMID: 2719343

“Focal fibromuscular dysplasia of small coronary arteries is not so rare as it is unrecognized.”
Morphologic characteristics and functional significance of focal fibromuscular dysplasia of small coronary arteries. Am J Cardiol. 1990 Apr 3;65(14):12G-22G. James TN. World Health Organization Cardiovascular Center, Galveston, Texas. PMID: 2181850

FMD eventually leads to death from heart attack, cancer, stroke or less commonly, from kidney failure, congestive heart failure, dissected arteries or burst aneurysms.

* The causes of death in patients with carotid FMD were heart attack (44.4%), cancer (33.3%) and stroke (22.2%) – now the three leading causes of death in the USA, in that order.
“Fibromuscular dysplasia of the internal carotid artery: long-term surgical results.” J Cardiovasc Surg (Torino). 1993 Dec;34(6):465-72. Moreau P, Albat B, Thevenet A. Service de Chirurgie Thoracique et Cardio-Vasculaire, Centre Hospitalier Universitaire, Montpellier, France. PMID: 8300709

Cancer, heart disease and stroke are the leading causes of death in the USA today. 1 in 2 American men will get cancer; one in 3 women. Once considered “diseases of civilization,” these 'diseases' are now epidemic around the world. The trends were identified in the early 1990’s, and responded to in part by the establishment of the US Environmental Genome Project (EGP) in 1997.

“Trends for mortality from heart disease, stroke, lung cancer, and, less distinctly, cancer other than lung cancer, tend to be similar in different countries, ... suggesting the existence of common causes amenable to the same preventive measures.”
Circulation. 1994 Jul;90(1):574-82. Heart disease, cancer, and stroke mortality trends and their interrelations. An international perspective. Thom TJ, Epstein FH. Epidemiology and Biometry Program, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, Md. PMID: 8026045
circ.ahajournals.org...;90/1/574

FMD is seldom diagnosed before a life threatening “event” occurs, and 65% of reported cases in the USA are diagnosed in autopsy, with an incidence rate of 1.1% in adults.

www.emedicine.com...

“FMD frequency in the USA: incidence of new cases in adults diagnosed by angiography – 0.6%; diagnosed in autopsy – 1.1%.”

NOTE: Incidence means new cases found yearly, presented as a % of the total population.
Also see Puri, PMID: 10334397.

Here's a table that puts together the stats for FMD incidence in autopsy with reported death stats:

FMD INCIDENCE in the USA

YEAR - DEATHS - TOTAL POP - EST ADULT POP (75%) - 1.1% ADULT POP:
FMD incid in autopsy

1999 - 2,391,399 - 279,295,000 - 209,471,250 - 2,304,184
1998 - 2,337,256 - 276,115,000 - 207,086,250 - 2,277,949
1997 - 2,314,245 - 272,912,000 - 204,684,000 - 2,251,524
1996 - 2,314,690 - 269,667,000 - 202,250,250 - 2,224,750
1995 - 2,312,132 - 266,557,000 - 199,917,750 - 2,199,095

Source: Population: Census; Reported Deaths; World Health Organization.

As you can see, nearly all of the reported deaths also are diagnosed with FMD in autopsy – so the obvious conclusion is that nearly 100% of the American population is infected with FMD by adulthood.




.

Originally posted by LazarusTheLong
The prion plague in perhaps a problem... but it only serves the need for culling the herd, so it is more of a solution than a problem...

...I would think they are using this as a method of herd culling... (your point) but is that bad?

Yes it's bad - because the elite are choosing the survivors. They are reserving diagnosis and treatment for themselves, and for 'designated support servants.'

...I happen not to respect thei choices - plus, I believe in democracy.

clarify if you would sofi:
how is this prion disease different from heart disease?
the effects seem to be the same, and the death stats seem to mimic each other...
could it be that heart disease is the prion disease, and we have known about it by another name all this time... and just now realize it is what heart disease is...?

It may not be responsible for all heart disease - but is certainly behind the current epidemics, and its spread to third world nations. See above post, ie:

For example:

“Fibromuscular dysplasia in a child: a generalized arterial disease.” Acta Paediatr Scand 1980 Jul;69(4):563-6. Pesonen E, Koskimies O, Rapola J, Jaakelainen J. PMID: 7446106

“Fibromuscular dysplasia may be a systemic disease…, and may affect the central nervous system.”
Renovascular hypertension and demyelimating disease in a young woman. Alaska Med 1989 Jul-Dec;31(4):137-43. Neubauer RL. PMID: 2596648

“This report suggests that FMD should be considered to be a systemic angiopathy…”
“Clinical significance of cerebral aneurysm in renovascular hypertension due to fibromuscular dysplasia: two cases in siblings.” Angiology 1989 Jun;40 (6):581-8. Ouchi Y, Tagawa H, Yamakado M, Takanashi R, Tanaka S. Department of Internal Medicine, Mitsui Memorial Hospital, Tokyo, Japan. PMID: 2719343

“Focal fibromuscular dysplasia of small coronary arteries is not so rare as it is unrecognized.”
Morphologic characteristics and functional significance of focal fibromuscular dysplasia of small coronary arteries. Am J Cardiol. 1990 Apr 3;65(14):12G-22G. James TN. World Health Organization Cardiovascular Center, Galveston, Texas. PMID: 2181850

FMD eventually leads to death from heart attack, cancer, stroke or less commonly, from kidney failure, congestive heart failure, dissected arteries or burst aneurysms.

* The causes of death in patients with carotid FMD were heart attack (44.4%), cancer (33.3%) and stroke (22.2%) – now the three leading causes of death in the USA, in that order.
“Fibromuscular dysplasia of the internal carotid artery: long-term surgical results.” J Cardiovasc Surg (Torino). 1993 Dec;34(6):465-72. Moreau P, Albat B, Thevenet A. Service de Chirurgie Thoracique et Cardio-Vasculaire, Centre Hospitalier Universitaire, Montpellier, France. PMID: 8300709

Cancer, heart disease and stroke are the leading causes of death in the USA today. 1 in 2 American men will get cancer; one in 3 women. Once considered “diseases of civilization,” these 'diseases' are now epidemic around the world. The trends were identified in the early 1990’s, and responded to in part by the establishment of the US Environmental Genome Project (EGP) in 1997.

“Trends for mortality from heart disease, stroke, lung cancer, and, less distinctly, cancer other than lung cancer, tend to be similar in different countries, ... suggesting the existence of common causes amenable to the same preventive measures.”
Circulation. 1994 Jul;90(1):574-82. Heart disease, cancer, and stroke mortality trends and their interrelations. An international perspective. Thom TJ, Epstein FH. Epidemiology and Biometry Program, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, Md. PMID: 8026045
circ.ahajournals.org...;90/1/574

FMD is seldom diagnosed before a life threatening “event” occurs, and 65% of reported cases in the USA are diagnosed in autopsy, with an incidence rate of 1.1% in adults.

www.emedicine.com...

“FMD frequency in the USA: incidence of new cases in adults diagnosed by angiography – 0.6%; diagnosed in autopsy – 1.1%.”

NOTE: Incidence means new cases found yearly, presented as a % of the total population.
Also see Puri, PMID: 10334397.

Here's a table that puts together the stats for FMD incidence in autopsy with reported death stats:

FMD INCIDENCE in the USA

YEAR - DEATHS - TOTAL POP - EST ADULT POP (75%) - 1.1% ADULT POP:
FMD incid in autopsy

1999 - 2,391,399 - 279,295,000 - 209,471,250 - 2,304,184
1998 - 2,337,256 - 276,115,000 - 207,086,250 - 2,277,949
1997 - 2,314,245 - 272,912,000 - 204,684,000 - 2,251,524
1996 - 2,314,690 - 269,667,000 - 202,250,250 - 2,224,750
1995 - 2,312,132 - 266,557,000 - 199,917,750 - 2,199,095

Source: Population: Census; Reported Deaths; World Health Organization.

As you can see, nearly all of the reported deaths also are diagnosed with FMD in autopsy – so the obvious conclusion is that nearly 100% of the American population is infected with FMD by adulthood.

same is true in africa... the civilized world has left the large part of the continent to its own destuction for decades... this is because they had hoped that it would solve its own problems with war famine and disease...

Corporate industry and resource harvesting created those problems - and didn't leave the poor Africans even iwth fresh water...

in another thread, i pointed out that science does have a cure, but can't use it until the population of the world is stable...(and nothing will be done before that)

IMO - the epidemic is creating population instability - we are hard-wired to reproduce when the species is threatened.

in fact... if a prion disease was rampant in the population... it would be a good thing for mother earth... and our children...(the ones arising from immune couples)...

much like many of the longevity medicines will coincidentally be released when the population can handle the stress...

facts are facts...
this world ain't big enough for all of us, and our health...

Again, it's not people who are destroying the earth's eco-systems - it's industry, and resource harvesting.

You are choosing profits over people.


The question I asked in the article was:


Should the human population be controlled to support the system, or should the system be changed to support life?


You have given us your answer.



.

Originally posted by Ghaleon4
Soficrow:
You're scaring the SH-T out of everyone here man...seriously...you've done all this research, can you PLEASE give us some good news!?
SHEESH...
What can we do already?!

Sorry all - tripped over a hot new prion story this morning - kept me busy - will post it tomorrow. ...This is a quick and dirty response - more later...

What can we do?


There are diagnostic tests, treatments and vaccines - which are not covered by insurance - and not available to ordinary people. Demand them, together. Don't roll over.

* “Blood test for prions?” 23 November 2000 www.nature.com...
* “Antibodies could combat prion-based diseases” August 2, 2001 www.biomedcentral.com...
* “Antibodies cripple prions.” 6 March 2003 www.nature.com...
* “Scientific Advance Establishes 'Proof Of Principle' That Prion Diseases Might Be Prevented” 6 March 2003, Nature. www.sciencedaily.com...
* “Researchers Discover Possible Diagnosis, Treatment, Vaccine For Mad Cow, Prion Diseases” 2003-06-02
www.sciencedaily.com...

Demand massive testing and access to treatments and vaccines. Demand clean ups, and effective sterilization and decontamination of hospitals and food production factories, to start. Demand that chemicals and toxins be removed from our food and the environment because they trigger the diseases and make them progress. DEMAND PREVENTION.

Demand that Environmental Protection Agency (EPA) regulations and controls be up-graded and implemented. There are nano-filters to clean them out of wastewater and drinking water - also not being used. Demand that they be installed and used.

Again. The technology does exist to kill these suckers. It's just not used. Implementing the new effective measures will cost, and no one is doing it voluntarily.


smallpeeps asked for a reference on how competing strains neutralize each other. Have thousands of files, can't find the specific reference I was looking for, but...

Here's a a quick outline of the 'competing strains' concept, one preliminary reference to the phenomeneon in prions, and a quick overview of self-help prion defense.

CONCEPT: when you have 2 strains of the same disease, in the absence of co or super-infection (eg., Nowak & May 1994), they both are competing for the same "prey" (eg., Armstrong & McGehee 1980). ...Prions prey on proteins of a similar shape to themselves - and make them morph into their prion shape on contact - they get their identity from their shape. If you have 2 or more prion strains in the same cell, they just keep changing each other back and forth, and the normal protein has a better chance for survival.

...although a trend exists for the risk of infection with prion disease to increase with repeated doses, it does so to a lesser degree than is expected if challenges combine independently or in a cumulative manner.
Proc Natl Acad Sci U S A. 2003 Sep 16;100(19):10960-5. Epub 2003 Sep 05. Repeated challenge with prion disease: the risk of infection and impact on incubation period. Gravenor MB, Stallard N, Curnow R, McLean AR. Institute for Animal Health, Compton, Berkshire RG20 7NN, United Kingdom. m.b.gravenor@swansea.ac.uk PMID: 12960400

SELF-HELP PRION DEFENSE


The curcumin in CURRY inhibits prion replication.

* J Virol. 2003 May;77(9):5499-502. Inhibition of protease-resistant prion protein accumulation in vitro by curcumin. Caughey B, Raymond LD, Raymond GJ, Maxson L, Silveira J, Baron GS. Laboratory of Persistent Viral Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Rocky Mountain Laboratories, Hamilton, Montana 59840, USA. bcaughey@nih.gov PMID: 12692251
jvi.asm.org...
* J Neurosci. 2001 Nov 1;21(21):8370-7. The curry spice curcumin reduces oxidative damage and amyloid pathology in an Alzheimer transgenic mouse. Lim GP, Chu T, Yang F, Beech W, Frautschy SA, Cole GM. Departments of Medicine and Neurology, University of California, Los Angeles, Los Angeles, California 90095, USA. PMID: 11606625
www.jneurosci.org...

These results suggest that frequent consumption of GREEN TEA enables the body to maintain a high level of tea polyphenols and this paper is the first pharmacological evidence of a wide distribution of [3H]EGCG in mouse organs, indicating a similar wide range of target organs for cancer prevention in humans.
Carcinogenesis. 1998 Oct;19(10):1771-6. Wide distribution of [3H](-)-epigallocatechin gallate, a cancer preventive tea polyphenol, in mouse tissue. Suganuma M, Okabe S, Oniyama M, Tada Y, Ito H, Fujiki H. Saitama Cancer Center Research Institute, Japan. masami@saitama-cc.go.jp PMID: 9806157

Several classes of compounds were represented in the 17 most potent inhibitors, including naturally occurring polyphenols (e.g., tannic acid and tea extracts), phenothiazines, antihistamines, statins, and antimalarial compounds. ...The fact that many are either approved human drugs or edible natural products should facilitate their use in animal testing and clinical trials.

* J Virol. 2003 Oct;77(19):10288-94. New inhibitors of scrapie-associated prion protein formation in a library of 2000 drugs and natural products. Kocisko DA, Baron GS, Rubenstein R, Chen J, Kuizon S, Caughey B. Laboratory of Persistent Viral Diseases, Rocky Mountain Laboratories, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Hamilton, Montana 59840, USA. PMID: 12970413
An alphabetical list of the compounds is available at the MicroSource Discovery website at www.msdiscovery.com/spect.html.
NOTE: Antihistamines. The antihistamines astemizole and terfenadine were both among the most potent PrPSc inhibitors. These compounds are known to be poor at crossing the blood-brain barrier, a fact which may limit their therapeutic usefulness against TSEs. These antihistamines have been used extensively in humans but are currently not marketed in the United States because of a concern for serious, but rare, cardiovascular toxicity and the availability of safer alternatives.

...eat a varied diet to prevent a concentration of one strain in the body.




PS. Smallpeeps the research constraints come thru defining prions as 'select agents' - and restricting research for "security" reasons. ...a joke, because they're everywhere already - and no terrorist is going to wait 20-odd years for a payoff.


.


.

[edit on 2-2-2005 by soficrow]

I've always liked Canadian scientist and environmental activist David Suzuki's example of just how fatal our situation has become, and something to think about for all those proposing the collonization of space as a possible solution:

...image a test tube full of food for bacteria to last 60 minutes, and imagine that we introduce a single bacterium that divides each minute. Because the population doubles every minute, you get exponential population growth. At 60 minutes, the test tube is full of bacteria and all the food is gone. Now here’s the interesting part: the test tube is 50% full at *59 MINUTES*, 25% full at *58 MINUTES*… 3% full at 55 minutes …

So Suzuki continues and proposes: Perhaps one of the bacterium figure out they’re going to run out of food at 55 minutes (3% full). So if the bacteria use all their resources to create another 3 test tubes of food, you’d think they would be saved wouldn’t they? Well, at 60 minutes the first test tube would be full of bacteria. At 61 minutes, the second test tube would be full of bacteria. At 62 minutes, all four test tubes would be full of bacteria. So they only bought themselves 2 more minutes of time even though they quadrupled their food.

The point of all this? Most scientists believe that we are well into the 59th minute.

The clock is ticking...

One way to help in prion research is to become a member of the World Communinty Grid and let your computer analyse data for The Human Proteome Folding Project. It's the same kind of setup as SETI@Home only vastly more important!!! Sign up now:

www.worldcommunitygrid.org...

-raven

Originally posted by Kidfinger
Sofi. Have you done any research on protien enzymes? Scientist have used a special enzime that attaches to DNA and severs it at a certain spot rendering it effectivly usless. Apparently it is used alot in recumbanant DNA research, and has the potential to be the virus killer of all virus killers. There are different forms of this enzyme. They all cut the DNA, they each just do it in a different place.

...enzymes that cut DNA don't touch prions because prions have no DNA. But new enzymes have been found that do kill prions...

Some quicks refs:

“…prions are notoriously difficult to break down… A new disinfectant, based on enzymes collected from a volcanic pool, is showing promise in destroying the mutated prion proteins…

“We don't know why prions are so highly stable, but they are extremely hard to destroy. Indeed, one standard method of decontamination – soaking in fomaldehyde – actually stabilises the prions.”

* “Volcanic pool enzyme kills prions” 29 July 03
www.newscientist.com...

* “Scientists Discover That Enzyme Degrades Mad Cow Disease Prion,” North Carolina State University press release, January 5, 2004. J. Shih.
www.ncsu.edu...


Novel methods for disinfection of prion-contaminated medical devices. Lancet. 2004 Aug 7;364(9433):521-6. Fichet G, Comoy E, Duval C, Antloga K, Dehen C, Charbonnier A, McDonnell G, Brown P, Lasmezas CI, Deslys JP. CEA/DSV/DRM/GIDTIP, Route du Panorama, 92265 Fontenay-aux-Roses, France.
ncbi

Mechanism of inactivation on prion conversion of the Saccharomyces cerevisiae Ure2 protein. Proc Natl Acad Sci U S A. 2002 Apr 16;99(8):5253-60. Baxa U, Speransky V, Steven AC, Wickner RB. Laboratory of Structural Biology, National Institute of Arthritis, Musculoskeletal, and Skin Diseases, National Institutes of Health, Bethesda, MD 20892, USA. PMID: 11959975
www.pnas.org...

New inhibitors of scrapie-associated prion protein formation in a library of 2000 drugs and natural products. J Virol. 2003 Oct;77(19):10288-94. Kocisko DA, Baron GS, Rubenstein R, Chen J, Kuizon S, Caughey B. Laboratory of Persistent Viral Diseases, Rocky Mountain Laboratories, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Hamilton, Montana 59840, USA. PMID: 12970413
jvi.asm.org...

Also - someone asked about heart disease and infectious prions earlier. Here's a quick connect-the-dots puzzle...

"Mitochondrial oxidative stress has been implicated in heart disease including myocardial preconditioning, ischemia/reperfusion, and other pathologies. In addition, oxidative stress in the mitochondria is associated with the pathogenesis of Alzheimer's disease, Parkinson's disease, prion diseases, and amyotrophic lateral sclerosis (ALS) as well as aging itself."
Applied proteomics: mitochondrial proteins and effect on function. Circ Res. 2002 Mar 8;90(4):380-9. Lopez MF, Melov S. Proteome Systems, Woburn, MA 01801, USA. mary.lopez@proteomesystems.com PMID: 11884366
circres.ahajournals.org...

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