Originally posted by FredT
I am very suspicious of the communist government in China. For one, both the initial outbreak of bird flu as well as SARS occurred in regions of China
that have been rumored to house biowarfare labs. Also given their initial cover-ups and the jailing of the doctor that alerted the world, something
smells fishy.
What's fishy are recurring attempts to blame bird flu and SARS on bioterrorism. I predicted this gambit long ago - and it's pure spin.
...Epidemiologists, virologists and other scientists have been warning authorities for over 35 years that this was coming - no bioterrorism
required.
FYI - the World Health Organization refuted the claim that bird flu originated in China, in January of 2004.
"WHO refutes claims that bird flu virus originated from China"
www.china.org.cn...
english.people.com.cn...
insidecostarica.com...
From Scientific American re SARS: "...scientists report that their findings support the idea that the SARS virus made the jump from animals to
humans, probably multiple times."
www.sciam.com...
More to the evolutionary point -
Bird flu and SARS are linked to a disease called fibromuscular dysplasia (FMD). FMD first appeared as a very rare
disease in the early 1900’s, in humans in the USA. It was acknowledged officially in 1938 when Drs. Leadbetter and Burkland proved it could be
transmitted congenitally and also, cause birth defects.
[Leadbetter WF: Hypertension in unilateral renal disease. J Urol 1938, 39:611-626.]
By 1980, Dr. L. M. Julian discovered FMD in domestic turkeys and in 1981, Drs. House and Benditt described FMD pathology in steelhead trout, both in
the USA. By 1996, FMD had spread to quail Japan. In 1999, the University of Texas Medical Branch reported that a secondary effect of FMD called
“angiomatosis” had spread to infect 100% of juvenile and adult Atlantic bottlenose dolphins since it was first recognized in 1992. FMD pathology
is found in SARS and avian flu, and in new mutated forms of other diseases.
Since 1938, it has become clear that fibromuscular dysplasia not only uses infectious microbes as vehicles of transmission but also, as mechanisms to
create new disease mutations. Radiation and other environmental contaminations were identified as significant factors that intensified disease
progression as well as cellular, genetic and microbial mutation. The mutation process was recognized as a cycle involving the disease bouncing back
and forth between people and animals, mainly via carrier microbes. The mutation cycle escalated dramatically between the 1970’s and 1990’s. By
2000, disease mutations that once took decades and generations to develop could appear in a single season.
The FMD epidemic and its related mutations were hidden from the public to protect international profits in insurance, the food and water industries,
and other polluting industries. Now it's time to pay the piper.
So if you want truth - not spin or faith-based science - then look to the early 1900's in the USA to find the origins of SARS and avian flu.
REFERENCE HIGHLIGHTS re avian flu
1975 - related re: mechanics FMD-virus cooperation "...alpha-actinin may be involved in the organization of actin filament bundles, in the attachment
of actin filaments to the plasma membrane, and in the assembly of actin filaments in areas of cell to cell contact." - PMID: 802682
1980 - domestic turkeys, USA - fibromuscular dysplasia, fibromuscular hyperplasia, and medial hyperplasia affects arterioles and small arteries, with
adherent thrombus - says "Angiopathy appears to be basic to the pathogenesis of fibromuscular dysplasia and is characterized by endothelial
hyperplasia, smooth-muscle vacuolization, and patchy necrosis of the media." - claims is first time disease found in animals - PMID: 7435545
1981 - FMD pathology in steelhead trout - Am J Pathol. 1981 Sep;104(3):250-7. The ultrastructure of spontaneous coronary arterial lesions in steelhead
trout (Salmo gairdneri). House EW, Benditt EP. PMID: 7294154
1986 - proteinaceous capsids using viruses as vehicles of transmission, RNA interference silences genes - Schmidt FR, Lemke PA, Esser K (1986) Viral
influences on aflatoxin formation by Aspergillus flavus. Appl Microbiol Biotechnol 24:248-252
1990 – Efficient incorporation of human CD4 protein into avian leukosis virus particles. PMID: 2175047
[Also see: 2002 - J Virol. 2002 Aug;76(15):7860-2. 1917 avian influenza virus sequences suggest that the 1918 pandemic virus did not acquire its
hemagglutinin directly from birds. Fanning TG, Slemons RD, Reid AH, Janczewski TA, Dean J, Taubenberger JK. Division of Molecular Pathology,
Department of Cellular Pathology and Genetics, Armed Forces Institute of Pathology, Rockville, Maryland 20850-3125, USA. PMID: 12097598]
1996 – FMD in quail, Japan – PMID: 8920213
1999 - FMD pathology in dolphins - Vet Pathol. 1999 Jan;36(1):28-34. Angiomatosis, a newly recognized disease in Atlantic bottlenose dolphins
(Tursiops truncatus) from the Gulf of Mexico. Turnbull BS, Cowan DF. Department of Pathology, University of Texas Medical Branch, Texas Marine Mammal
Stranding Network, Galveston 77555-0588, USA. PMID: 9921753
2001 – avian reovirus – FMD pathology in infected chickens, Brazil
www.scielo.br...
2003 - bypassing immune system - on antigen presenting cells, such as B cells and fibroblasts, (cytoskeletal) actin microfilaments act as a regulator
of the movement and capping of HLA-DR receptors. PMID: 12590977
Refrences from the US National Institutes of Health are filed by "PMID #'s" - go to
www.ncbi.nlm.nih.gov... and enter the
PMID # in the search box to read the abstract.
.
[edit on 10-1-2005 by soficrow]