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originally posted by: ServantOfTheLamb
Cells preform an action during meiotic division called Homologus recombination. Homologus recombination can either between genes which leaves preexisting genes untouched, and Homologus recombination in genes which can result in the formation of new alleles.
We detected XMRV infection in the two cell lines and in the later passage xenografts, but not in the early passages. In particular, we found that the host mice contained two proviruses, PreXMRV-1 and PreXMRV-2, which share 99.92% identity with XMRV over >3.2-kilobase stretches of their genomes. We conclude that XMRV was not present in the original CWR22 tumor but was generated by recombination of two proviruses during tumor passaging in mice. The probability that an identical recombinant was generated independently is negligible (~10−12); our results suggest that the association of XMRV with human disease is due to contamination of human samples with virus originating from this recombination event.
We are hosts to many organisms, Bacteria and viruses that mutated and became PHAGES. Such as the white blood cell is an ancient linage of intergration between bacteria and virus to form a phage of which has a symbiotic relationship.
You try to say that mutation does not account for the new imputes of data. How does a back person gain the color of blue eyes, Just by randomly assorting already present allels?
The common ancestor thing is kinda unavoidable when pathogens are everywhere and outnumber us by the trillions.
Your gut is inhabitted by bacteria that break down food into enzymes that the cellulars can absorb. So even then, The bacteria that enters our body influences our DNA and overall what makes us US. with a ratio of 10 bacteria to 1 human cell.
The picture is a lot more complicated then matching capital and miniscule letters in a 4 box grid and having that explain diversity. The traits have to be inherited through pathogens first before they even reach the populations geonome!
Are you suggesting that there is intelligent changes/interference occurring at, or between, recombination stages? If so, what intelligence is causing the "interference"?
originally posted by: mikefougnie
a reply to: ServantOfTheLamb
You have just described Natural Selection which leads to changes within species of animals through loss or mutation of existing genetic material. This does not account for the initial data.
All my point was is, There are many *allells* and even differnt proteins some people lack all together *except for the CORE template of course*. The blue eye trait was inherited by from a Virus. Diversity changes, As the micro life forms change we change along with them.
evs can be constantly written in the more *inactive* parts of the DNA. Consuming different microbes would undoubtly also influence diet and not by chance. Physical features to better suit that diet. Such as the Darwin finches.
my comment about the comman ancestor is pathogens roam around freely in the outside world. It's nearly impossible to tell the difference if humans contracted the traits externally or from * A genetic ancestor* through breeding. So breeding is not completely a viable solution in how these traits come to arise.
But many of these new traits that are picked up were from enviromental factors rather than soely reproduction. All reproduction is carry those traits, it does not invent them.
but to say that mutation is not a driving factor in it is not true because the source of these alleles in the first place came from DNA exchange through micro(Vampirism) lol.